CALL FOR PAPERS Angiogenesis in Health and Disease Adenoviral PR39 improves blood flow and myocardial function in a pig model of chronic myocardial ischemia by enhancing collateral formation

نویسندگان

  • Mark J. Post
  • Kaori Sato
  • Masahiro Murakami
  • Jialin Bao
  • Daniela Tirziu
  • Justin D. Pearlman
  • Michael Simons
چکیده

Mark J. Post, Kaori Sato, Masahiro Murakami, Jialin Bao, Daniela Tirziu, Justin D. Pearlman, and Michael Simons Angiogenesis Research Center and Section of Cardiology, Dartmouth Medical School, Lebanon, New Hampshire; Beth Israel Deaconess Medical Center, Boston, Massachusetts; and Department of Physiology, Cardiovascular Research Institute Maastricht, Maastricht University, Maastricht, The Netherlands

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Adenoviral PR39 improves blood flow and myocardial function in a pig model of chronic myocardial ischemia by enhancing collateral formation.

Angiogenic therapy with individual growth factors or "master switch" genes is being evaluated for treatment of advanced coronary artery disease. In this study, we investigated the efficacy and mechanism of PR39, a gene capable of activating VEGF and fibroblast growth factor (FGF)-2-dependent pathways. PR39 enhances hypoxia-inducible factor-1alpha (HIF-1alpha)-dependent gene expression by select...

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Efficacy of intracoronary versus intravenous FGF-2 in a pig model of chronic myocardial ischemia.

BACKGROUND Therapeutic angiogenesis in ischemic myocardium has been shown to be a feasible and effective strategy to improve regional blood flow and myocardial function. However, the optimal mode of growth factor administration still needs to be established. METHODS Using a pig model of chronic myocardial ischemia, we evaluated the efficacy of intravenous and intracoronary infusion of FGF-2 a...

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تاریخ انتشار 2006