Advances in Brief p14 Modulates the Cytolytic Effect of ONYX-015 in Mesothelioma Cells with Wild-type p53

نویسندگان

  • Cheng-Ta Yang
  • Liang You
  • Kazutsugu Uematsu
  • Che-Chung Yeh
  • Frank McCormick
  • David M. Jablons
چکیده

ONYX-015 has been reported to kill selectively tumor cells lacking functional p53. Genetic alterations of INK4a/ARF locus, which is a predominant event in malignant pleural mesothelioma, may result in loss of p14 and subsequent disruption of p53 pathway in cancer cells. In the present study, ONYX-015 was able to kill three mesothelioma cell lines (H28, H513, and 211H) with wild-type p53 but lacking p14. In contrast, MS-1 mesothelioma cells, which expressed both p53 and p14, were resistant to ONYX-015. Introducing p14 gene into the H28 cell, a mesothelioma cell without p14 expression, significantly increased the resistance of this cell line to the cytolytic effect of ONYX-015. Our results suggest that human mesotheliomas with wild-type p53 yet lacking p14 are potential candidates for ONYX-015 therapy.

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p14(ARF) modulates the cytolytic effect of ONYX-015 in mesothelioma cells with wild-type p53.

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تاریخ انتشار 2001