Prevention of Contrast-Induced Nephropathy with the Combination of Sodium Bicarbonate and N-Acetylcysteine: A Randomized Controlled Trial

نویسنده

  • Rebecca L. Toonkel
چکیده

Following coronary angiography, approximately 14.5% of patients develop contrast induced nephropathy (CIN). In addition, CIN accounts for more than 10% of hospital acquired renal failure. Not only is CIN associated with increased lengths of stay, but also with worsening of baseline renal function, and, rarely, progression to hemodialysis. Most importantly, CIN is associated with increased short and long term mortality. Several studies have shown that the risk factors for developing CIN include baseline renal impairment, diabetes mellitus, and larger contrast loads (as with coronary angiography). While the exact pathophysiology of CIN is not known, it is thought that iodinated radiographic contrast causes direct tubular toxicity via free radical formation (which is promoted by the acidic tubular environment), and by tubular vasoconstriction leading to ischemic injury. Based on these insights, many attempts have been made to prevent CIN. It is now well established that the use of low osmolarity, non-ionic contrast agents is associated with decreased risk of CIN. In addition, good evidence supports the use of the free radical scavenger N-Acetylcysteine (NAC) for the prevention of CIN. Perhaps most widely accepted, intravenous hydration is considered integral for the prevention of CIN. In this setting, normal saline is the most frequently used intravenous fluid as it was shown in one study to be more effective than half-normal saline. Most recently, however, new evidence suggests that hydration with sodium bicarbonate is more effective for prevention of CIN than hydration with normal saline. This study is designed to determine if there is a role for sodium bicarbonate in addition to NAC in the prevention of CIN in patients with renal insufficiency undergoing coronary angiography.

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تاریخ انتشار 2006