Renal Oxygen Consumption in Man during Abdominal Compression.

The renal blood flow, glomerular filtration rate and maximal rates of glucose reabsorption and diedrast excretion are greatly reduced in man by increased intra-abdominal pressure (1). These effects may be attributed to simultaneous elevations in renal venous pressure and intra-pelvic pressure which act respectively to decrease blood flow and to halt urine flow from nephrons which have relatively low terminal intra-luminal pressures. Thus a large proportion of the renal parenchyma apparently no longer functions in producing urine. It seemed of interest to determine whether renal oxygen consumption is altered under these conditions.