Vitamin E deficiency in the rat
نویسندگان
چکیده
منابع مشابه
Vitamin E deficiency and ion transport in rat-liver slices.
Rat-liver slices lose potassiumwhenplaced in cold saline solution and reaccumulate potassium when placed in warm oxygenated Ringer solution (McLean, 1963). Weanling rats fed on a yeast diet which is deficient in vitamin E and selenium suffer acute liver necrosis at 17 ± 4 days after starting to eat the diet. Liver slices taken from rats given this diet for 7 days or more cannot reaccumulate pot...
متن کاملVitamin E Deficiency in the Monkey . Iv .
NEMIA regularly develops in the rhesus monkey (Macaca mulatta) when it is deprived of vitamin E.1 The anemia is accompanied by granulocytosis,’ an increased concentration of bone marrow deoxyribonucleic acid,2 a reduction in the maximal Cr5’ survival time of the erythrocytes,3 a slightly elevated reticulocyte count1 and a prompt reticulocytosis following vitamin E administration. This combinati...
متن کاملVitamin E Deficiency in the Monkey
Eight young rhesus monkeys were fed a purified diet devoid of vitamin E. After from 6 to 13 months of feeding, all the animals developed signs of vitamin E deficiency. The signs of vitamin E deficiency in the monkey include muscular dystrophy, elevated excretion of creatine, allantoin, and free amino acids and decreased excretion of creatinine. The vitamin E-deficient monkeys all developed anem...
متن کاملEffect of selenium deficiency and vitamin E deficiency on glutathione metabolism in isolated rat hepatocytes.
Selenium deficiency and vitamin E deficiency both affect xenobiotic metabolism and toxicity. In addition, selenium deficiency causes changes in the activity of some glutathione-requiring enzymes. We have studied glutathione metabolism in isolated hepatocytes from selenium-deficient, vitamin E-deficient, and control rats. Cell viability, as measured by trypan blue exclusion, was comparable for a...
متن کاملAtaxia with Vitamin E Deficiency in Norway
OBJECTIVE Ataxia with vitamin E deficiency (AVED) is a rare autosomal recessive neurological disorder which usually starts in childhood. The clinical presentation is very similar to Friedreich ataxia, most patients have progressive truncal and extremity ataxia, areflexia, positive Babinski sign, dysarthria and sensory neuropathy. METHODS We made an inquiry to our colleagues in Norway, we incl...
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ژورنال
عنوان ژورنال: Biochemical Journal
سال: 1943
ISSN: 0306-3283
DOI: 10.1042/bj0370523