Transient Ischemic Attacks and Ischemic Stroke: Indications for Anticoagulation
نویسندگان
چکیده
منابع مشابه
Transient ischemic attacks: predictability of future ischemic stroke or transient ischemic attack events
The short-term risk of an ischemic stroke after a transient ischemic attack (TIA) is estimated to be approximately 3%-10% at 2 days, 5% at 7 days, and 9%-17% at 90 days, depending on active or passive ascertainment of ischemic stroke. Various risk prediction scores are available to identify high-risk patients. We present here a pragmatic review of the literature discussing the main scoring syst...
متن کاملTransient cerebral ischemic attacks.
“Stroke” is defined as rapid onset of focal neurological deficit, resulting from diseases of the cerebral vasculature and its contents. The term “transient ischemic attacks” (TIA) implies warning symptoms of stroke usually lasting upto 30 minutes to one hour, and complete recovery within 24 hours. Second TIA often causes more damage than the first. Without proper treatment one out of ten subjec...
متن کاملIschemic Preconditioning From Transient Ischemic Attacks?
Reduced impact of ischemia after an initial ischemic insult—ischemic preconditioning—has been demonstrated in a number of animal models. Transient ischemic attack (TIA) may produce ischemic preconditioning in people who have a subsequent stroke within days of an initial ischemic insult. We performed a cohort study of all 1707 patients given a diagnosis of TIA by an emergency department physicia...
متن کاملHeadache and transient ischemic attacks.
Headache and Transient Ischemic A Hacks • Headache occurring in association with episodes of transient cerebral ischemia has not been emphasized as part of the symptom complex. In an effort to ascertain the frequency and characteristics of these headaches and to determine whether there is any localizing, diagnostic, or prognostic significance to them, the records of 240 TIA patients were review...
متن کاملPathogenesis of Transient Ischemic Attacks and Stroke in Baboons
We describe a series of experiments in which a subhuman primate model was used to create temporary and permanent cerebral ischemia by three separate mechanisms. In the first group of five baboons, a hemodynamic model was produced by creating unilateral and bilateral carotid stenotic lesions of varying degrees with and without associated reduction in systemic perfusion pressure. Only global isch...
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ژورنال
عنوان ژورنال: Clinical Cardiology
سال: 1990
ISSN: 0160-9289
DOI: 10.1002/clc.1990.13.s6.29