The general anesthetic propofol increases brainN-arachidonylethanolamine (anandamide) content and inhibits fatty acid amide hydrolase

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The postmortal accumulation of brain N-arachidonylethanolamine (anandamide) is dependent upon fatty acid amide hydrolase activity.

N-arachidonylethanolamine (AEA) accumulates during brain injury and postmortem. Because fatty acid amide hydrolase (FAAH) regulates brain AEA content, the purpose of this study was to determine its role in the postmortal accumulation of AEA using FAAH null mice. As expected, AEA content in immediately frozen brain tissue was significantly greater in FAAH-deficient (FAAH-/-) than in wild-type mi...

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ACCELERATED COMMUNICATION Role of Fatty Acid Amide Hydrolase in the Transport of the Endogenous Cannabinoid Anandamide

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A Personal Retrospective: Elevating Anandamide (AEA) by Targeting Fatty Acid Amide Hydrolase (FAAH) and the Fatty Acid Binding Proteins (FABPs)

This perspective was adapted from a Career Achievement Award talk given at the International Cannabinoid Research Society Symposium in Bukovina, Poland on June 27, 2016. As a biochemist working in the neurosciences, I was always fascinated with neurotransmitter inactivation. In 1993 we identified an enzyme activity that breaks down anandamide. We called the enzyme anandamide amidase, now called...

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ژورنال

عنوان ژورنال: British Journal of Pharmacology

سال: 2003

ISSN: 0007-1188

DOI: 10.1038/sj.bjp.0705334