The common bacterial toxins hypothesis of sudden infant death syndrome

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منابع مشابه

Staphylococcal toxins and sudden infant death syndrome.

AIMS To investigate the hypothesis that commonly occurring bacterial toxins cause sudden infant death syndrome (SIDS) by (1), determining in which tissues bacterial toxins are concentrated after intravenous injection in rats; and (2), seeing if the same tissues contain detectable toxins in cases of SIDS. METHODS The tissue distribution of intravenously injected staphylococcal enterotoxin A (S...

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Mothers’ performance in prevention of Sudden Infant Death Syndrome

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Sudden Infant Death Syndrome and Sudden Unexpected Infant Death

Sudden infant death has haunted humanity since Biblical times, and many people are still confused about how to differentiate sudden infant death syndrome from other causes of sudden infant death, such as myocarditis or congenital heart disease. Because of the difficulty of diagnosis, the authors say that SIDS has become a “diagnostic dustbin,” but unnecessarily so. They provide the diagnostic c...

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The sudden infant death syndrome.

From the Department of Pathology, Chil­ dren’s Hospital, and Harvard Medical School — both in Boston (H.C.K.); and the Department of Pediatrics, Washing­ ton University School of Medicine, St. Louis (B.T.T.). Address reprint requests to Dr. Kinney at the Department of Pathol­ ogy, Enders Bldg. 1112, Children’s Hospi­ tal Boston, 300 Longwood Ave., Boston, MA 02115, or at hannah.kinney@childrens...

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The epidemiology of sudden infant death syndrome.

BACKGROUND Twins compared to singletons are at increased risk of sudden infant death syndrome (SIDS). AIMS To compare the epidemiology of SIDS in twins and singletons and to test the hypothesis that monozygous (MZ) were at greater risk of SIDS than dizygous (DZ) twins. METHODS Data from the Office for National Statistics on all registered live births and infant deaths with registered cause ...

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ژورنال

عنوان ژورنال: FEMS Immunology & Medical Microbiology

سال: 1999

ISSN: 0928-8244,1574-695X

DOI: 10.1111/j.1574-695x.1999.tb01322.x