Selective Glial Vulnerability following Transient Global Ischemia in Rat Brain
نویسندگان
چکیده
منابع مشابه
P55: Neuroprotective and Neurogenesis Effects of Curcumin in the Adult Rat Brain Following Transient Global Ischemia
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متن کاملReduction in ischemic brain injury following the administration of pentoxifylline after transient global ischemia/ reperfusion in a rat model
Background: It is well known that the hippocampus, the CA1 Pyramidal cells in particular, is selectively vulnerable during global cerebral ischemia. Recently, it is observed that pentoxifylline has a neuroprotective effect. This study explored the pharmacological relationship between ischemia-induced cell death of the hippocampus and the efficacy of a vasodilator agent (pentoxifylline) in the...
متن کاملp55: neuroprotective and neurogenesis effects of curcumin in the adult rat brain following transient global ischemia
traumatic brain injury resulting road accidents create damage to the brain. the severe brain injury may cause extensive tissue loss of several parenchyma which results in cavities due to primary destruction and secondary injuries such as ischemia and inflammation. recent findings suggest that neuronal precursors in the adult mammalian brain can be a therapeutic target in ischemic brain injuries...
متن کاملreduction in ischemic brain injury following the administration of pentoxifylline after transient global ischemia/ reperfusion in a rat model
background: it is well known that the hippocampus, the ca1 pyramidal cells in particular, is selectively vulnerable during global cerebral ischemia. recently, it is observed that pentoxifylline has a neuroprotective effect. this study explored the pharmacological relationship between ischemia-induced cell death of the hippocampus and the efficacy of a vasodilator agent (pentoxifylline) in the p...
متن کاملIncreased sensitivity to transient global ischemia in aging rat brain.
Transient global brain ischemia induced by cardiac arrest and resuscitation (CAR) results in reperfusion injury associated with oxidative stress. Oxidative stress is known to produce delayed selective neuronal cell loss and impairment of brainstem function, leading to post-resuscitation mortality. Levels of 4-hydroxy-2-nonenal (HNE) modified protein adducts, a marker of oxidative stress, was fo...
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ژورنال
عنوان ژورنال: Journal of Neuropathology & Experimental Neurology
سال: 1998
ISSN: 0022-3069,1554-6578
DOI: 10.1097/00005072-199803000-00004