Primary hyperparathyroidism presenting as torsades de pointes.
نویسندگان
چکیده
منابع مشابه
Torsades de Pointes (TdP) after Taking Prenylamine
L'intervalle de temps variable existante entre le debut du traitement par la prenylamine et la survenu de toisade de pointe et d'autre oart la raretee de· telle camplication est considere par les auteurs c0mme une indice de !'intervention de d'autres facteurs, parmie Jes quelles, la perturbation electrolytique doit jouer le role principal.
متن کاملMethadone-induced Torsades de pointes.
Torsades de pointes is a polymorphic ventricular tachycardia that can quickly evolve into ventricular fibrillation and sudden death. This arrhythmia often occurs secondary to medication- induced cardiac repolarization dysfunction with resultant prolonged QTc interval on ECG. Numerous medications can predispose patients to this deadly tachycardia. We report a case of methadone-induced Torsades d...
متن کاملTorsades de pointes: unanswered questions.
Torsade de pointes (Tdp) is a polymorphic ventricular tachycardia (VT) in which the axis of the QRS complex changes direction after a certain number of complexes as if the complex rotated around the baseline. Tdp is usually associated with QT prolongation, and dispersion of ventricular repolarlization (DR). Experimental models of tdp are usually associated with induction of early after depolari...
متن کاملPrimary hypothyroidism presenting with Torsades de pointes type tachycardia: a case report
BACKGROUND Hypothyroidism can manifest with cardiac abnormalities, often consisting of a combination of morphologic and functional changes. Low voltage, sinus bradycardia, and slowed conduction are usually found on electrocardiography.There are few reports of occurrence of torsades de pointes as the first presentation of long QT syndrome in the course of hypothyroidism. CASE PRESENTATION In p...
متن کاملSuppression of torsades de pointes by atropine.
A 67 year old woman with a history of chronic atrial fibrillation presented with asthma cardiale. She took no medication and there was no family history of long QT syndrome. She was treated with furosemide, nitroprusside, acenocoumarol, and digoxin. Two days later excessively prolonged RR intervals, which were terminated by escape beats with a right bundle branch block morphology, suggested imp...
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ژورنال
عنوان ژورنال: Heart
سال: 1993
ISSN: 1355-6037
DOI: 10.1136/hrt.70.5.473