Orthostatic stress, haemorrhage and a bankrupt cardiovascular system
نویسندگان
چکیده
منابع مشابه
Orthostatic hypotension following acute intracerebral haemorrhage.
BACKGROUND Blood pressure regulation may be impaired following acute stroke. Typically, there is overactivity of the sympathetic nervous system and underactivity of the parasympathetic system resulting in transient hypertension. Orthostatic hypotensive responses in acute stroke are less well documented. CASE REPORT We present a case of severe persistent orthostatic hypotension (OH) following ...
متن کاملComputational modeling of cardiovascular response to orthostatic stress.
The objective of this study is to develop a model of the cardiovascular system capable of simulating the short-term (< or = 5 min) transient and steady-state hemodynamic responses to head-up tilt and lower body negative pressure. The model consists of a closed-loop lumped-parameter representation of the circulation connected to set-point models of the arterial and cardiopulmonary baroreflexes. ...
متن کاملOrthostatic hypotension and cardiovascular risk.
Orthostatic hypotension (OH) results from a failure of neural and circulatory mechanisms to compensate for the reduction in venous return that normally occurs on assuming the upright posture. OH is defined as a fall in systolic blood pressure of 20 mm Hg or diastolic blood pressure of 10 mm Hg measured within 3 minutes of standing.1 OH can result from side effects of medications, intravascular ...
متن کاملA Model for Simulation of Infant Cardiovascular Response to Orthostatic Stress
We developed an infant circulation model which incorporates an accurate myocardial cell model including a beta adrenergic system. The beta adrenergic system is essential for the response reproduction of the baroreflex control system. The proposed model was constructed by modifying the parameters of a human adult circulation model with the aid of a guinea pig myocardial cell model, whose baselin...
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ژورنال
عنوان ژورنال: The Journal of Physiology
سال: 2009
ISSN: 0022-3751
DOI: 10.1113/jphysiol.2009.181495