Normal reproductive organ development in CF-1 mice following prenatal exposure to bisphenol A

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Normal reproductive organ development in CF-1 mice following prenatal exposure to bisphenol A.

Bisphenol A (BPA) is a monomer used in the manufacture of a multitude of chemical products, including epoxy resins and polycarbonate. The objective of this study was to evaluate the effects of BPA on male sexual development. This study, performed in CF-1 mice, was limited to the measurement of sex-organ weights, daily sperm production (DSP), epididymal sperm count, and testis histopathology in ...

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Normal Reproductive Organ Development

Bisphenol A (BPA) is a monomer used in the manufacture of a multitude of chemical products, including epoxy resins and polycarbonate. The objective of this study was to evaluate the effects of BPA on male sexual development. This study, performed in CF-1 mice, was limited to the measurement of sex organ weights, daily sperm production (DSP), epididymal sperm count, and testis histopathology in ...

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Exposure to bisphenol A, but not phthalates, increases spontaneous diabetes type 1 development in NOD mice

Type 1 diabetes mellitus (T1DM) is an autoimmune destruction of insulin producing pancreatic beta-cells due to a genetic predisposition and can be triggered by environmental factors. We have previously shown that bisphenol A (BPA) accelerates the spontaneous development of diabetes in non-obese diabetic (NOD) mice. Here, we hypothesized that oral exposure to a mixture of the endocrine disruptor...

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Transmaternal bisphenol A exposure accelerates diabetes type 1 development in NOD mice.

Diabetes mellitus type 1 is an autoimmune disease with a genetic predisposition that is triggered by environmental factors during early life. Epidemiological studies show that bisphenol A (BPA), an endocrine disruptor, has been detected in about 90% of all analyzed human urine samples. In this study, BPA was found to increase the severity of insulitis and the incidence of diabetes in female non...

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1 Human Exposure to Bisphenol a ( Bpa )

Acknowedgements: The authors gratefully acknowledge expertise and input from additional panel members: Jane C. Atkinson, Antonia M. Calafat, Frederick Eichmiller, Albert Kingman, Ruthann Rudel, and Kristina A. Thayer. This review was prepared in conjunction with the Bisphenol A Conference, Chapel Hill, NC, November 28-29, 2006. Support was provided by the National Institute of Environmental Hea...

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ژورنال

عنوان ژورنال: Toxicological Sciences

سال: 1999

ISSN: 1096-6080,1096-0929

DOI: 10.1093/toxsci/50.1.36