Narcolepsy and excessive daytime sleepiness

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Excessive daytime sleepiness.

Excessive daytime sleepiness is one of the most common sleep-related patient symptoms, and it affects an estimated 20 percent of the population. Persons with excessive daytime sleepiness are at risk of motor vehicle and work-related incidents, and have poorer health than comparable adults. The most common causes of excessive daytime sleepiness are sleep deprivation, obstructive sleep apnea, and...

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Pharmacotherapy for excessive daytime sleepiness.

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th17 cell related cytokine profiles in narcolepsy and other types of excessive daytime sleepiness

background and objective : narcolepsy is a chronic neurological disorder caused by loss of hypocretin (hcrt) neurons. both genetic and environmental factors play an important role for the development of narcolepsy. the mechanism of hcrt loss in narcolepsy is elusive; however, an autoimmune mediated destruction of hcrt neurons is suspected. the purpose of this study was to assess th17 related cy...

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Assessment and management of excessive daytime sleepiness.

The obstructive sleep apnoea/hypopnoea syndrome (OSAHS) is much the commonest ‘medical’ cause of sleepiness (Table 1), occurring in 1–4% of the middle-aged. OSAHS is the combination of sleepiness or two other major symptoms, such as unrefreshing sleep, difficulty concentrating and nocturnal choking, with five or more apnoeas plus hypopnoeas per hour slept (‘apnoea’ is defined as a 10-sec pause ...

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Introduction: narcolepsy and excessive daytime sleepiness: from the bench to the bedside.

arcolepsy is a chronic, neurologic sleep disorder resulting from the dysregulation of sleep-wake cyN cles. Narcoleptic patients experience excessive daytime sleepiness, cataplexy, sleep paralysis, and hypnagogic hallucinations. Narcolepsy is about as prevalent as multiple sclerosis, and can be as disabling in its consequences, yet it is underrecognized and underdiagnosed. Because (narcoleptic) ...

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ژورنال

عنوان ژورنال: BMJ

سال: 2004

ISSN: 0959-8138,1468-5833

DOI: 10.1136/bmj.329.7468.724