Maximum Principal Strain and Strain Rate Associated with Concussion Diagnosis Correlates with Changes in Corpus Callosum White Matter Indices
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چکیده
منابع مشابه
Regional axonal loss in the corpus callosum correlates with cerebral white matter lesion volume and distribution in multiple sclerosis.
Previous imaging studies have suggested that there is substantial axonal loss in the normal-appearing white matter (NAWM) of brains from multiple sclerosis patients and that this axonal loss may be an important determinant of disability. Recently, substantial axonal loss in the NAWM has been confirmed directly in post-mortem tissue. Whether the NAWM changes occur as a consequence of damage to a...
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Basal encephalocele is a rare craniofacial anomaly. In the present paper we report a 10-year-old boy presented with cleft palate, congenital nystagmus, and hypertelorism. During preoperative evaluation for cleft palate repair, a pulsatile mass was detected in the pharynx. Magnetic resonance imaging showed sphenoethmoidal type of basal encephalocele and agenesis of corpus callosum. Neurosurgical...
متن کاملOrganising white matter in a brain without corpus callosum fibres.
Isolated corpus callosum dysgenesis (CCD) is a congenital malformation which occurs during early development of the brain. In this study, we aimed to identify and describe its consequences beyond the lack of callosal fibres, on the morphology, microstructure and asymmetries of the main white matter bundles with diffusion imaging and fibre tractography. Seven children aged between 9 and 13 years...
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Introduction White matter fMRI is a controversial idea. To date, the majority of fMRI studies have focused on gray matter, largely for two reasons: 1) cerebral blood flow/volume is reduced in white matter relative to gray matter (e.g., [1, 2]); and 2) post-synaptic potentials are thought to be the primary source of the signal measured in fMRI (e.g., [3]). Despite this, a growing number of studi...
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ژورنال
عنوان ژورنال: Annals of Biomedical Engineering
سال: 2011
ISSN: 0090-6964,1573-9686
DOI: 10.1007/s10439-011-0402-6