Ischaemia induced QRS widening with acute inferior myocardial infarction
نویسندگان
چکیده
منابع مشابه
Transient QRS changes simulating acute myocardial infarction.
The purpose of this study was to determine the characteristics and incidence of abrupt occurrence of abnormal initial QRS forces that cannot be explained by acute myocardial infarction or left or right ventricular overload. Computerized data from 3175 patients with suspected acute infarction were reviewed to identify those in whom the ECGs revealed QRS complexes considered to be diagnostic (Q w...
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Wystąpienie ostrego niedokrwienia kończyny (ALI, acute limb ischaemia) wiąże się ze znacznym ograniczeniem lub zatrzymaniem przepływu w tętnicach kończyny dolnej. Jedną z przyczyn ALI może być zatorowość (35–40%) sercopochodna w przebiegu np. migotania przedsionków lub zawału serca (80%). Objawy ostrego niedokrwienia kończyn dolnych zależą od liczby i średnicy naczyń, które uległy całkowitemu z...
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On admission the physical examination was normal with a blood pressure of 130/90 mmHg. The initial electrocardiogram (Figure 1A) showed sinus rhythm with convex-upwards ST segment elevation (transmural injury) decreasing from V1 to V3 and acute inferior myocardial infarction pattern with Q waves and ST segment elevation of transmural injury in leads III and aVF. The patient was medicated with a...
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Twenty-six patients underwent arterial counterpulsation for refractory heart failure without shock complicating acute myocardial infarction. Patients were divided into a group of 12 with continuing myocardial ischaemia, evidenced by anginal pain associated with abnormal ST segment elevation, and a group of 14 without continuing ischaemia. Clinical features (apart from pain) and prognostic indic...
متن کاملQRS mapping in the evaluation of acute anterior myocardial infarction.
In 42 patients with acute anterior myocardial infarction (AMI), we studied the course of Q-wave development and R-wave reduction during the first 48 hours after the onset of chest pain. We used precordial mapping in relation to clinical features, hemodynamic measurements and enzyme release. Q waves developed within 6-14 hours (mean 9 hours) after onset of symptoms. R-wave amplitudes demonstrate...
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ژورنال
عنوان ژورنال: Heart
سال: 2005
ISSN: 1355-6037
DOI: 10.1136/hrt.2004.047670