Estimating afterload, systemic vascular resistance and pulmonary vascular resistance in an intensive care setting
نویسندگان
چکیده
منابع مشابه
Systemic vascular resistance: an unreliable index of left ventricular afterload.
Systemic vascular resistance (SVR) is a frequently used clinical index of left ventricular afterload. However, SVR may not adequately assess left ventricular afterload (i.e., ventricular internal fiber load during systole) since it reflects only peripheral vasomotor tone. In contrast, left ventricular end-systolic wall stress (sigma es) reflects the combined effects of peripheral loading condit...
متن کاملPulmonary vascular resistance.
where P represents the pressure gradient between the pulmonary artery and the pulmonary vein, F, the blood flow per unit time, and R. the vascular resistance. For a given pressure gradient and flow rate, R will be determined, among other things, by the physical dimensions of the chaniiels that the blood traverses. In a rigid hydraulic system this formula applies over a wide range of flow and pr...
متن کاملPulmonary Vascular Resistance
where P represents the pressure gradient between the pulmonary artery and the pulmonary vein, F, the blood flow per unit time, and R. the vascular resistance. For a given pressure gradient and flow rate, R will be determined, among other things, by the physical dimensions of the chaniiels that the blood traverses. In a rigid hydraulic system this formula applies over a wide range of flow and pr...
متن کاملPulmonary vascular resistance in emphysema.
To assess the hemodynamic effects of pulmonary microvasculature disruption in emphysema, we examined resting pulmonary hemodynamics and lung function in 12 carefully identified patients with type A chronic obstructive pulmonary disease. Individuals with respiratory muscle weakness and intercurrent infection were excluded. Standard spirometry, helium dilution lung volumes, and single-breath carb...
متن کاملSystemic vascular resistance should be banned.
In an article published in a recent issue of CHEST (June 2014), Marik 1 mentioned, “In patients with septic shock who are fl uid responders (an increase in cardiac output with fl uid boluses), vasodilatation with a fall in systematic vascular resistance has been observed.... Hence, although the cardiac output increases, vasodilatation occurs.” I believe that this statement is erroneous because ...
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ژورنال
عنوان ژورنال: IFAC Proceedings Volumes
سال: 2012
ISSN: 1474-6670
DOI: 10.3182/20120829-3-hu-2029.00112