Dialogue between Xop-T3SS Effectors of <I>Xanthomonas oryzae Pv. Iryzae</I> and Rice during Bacterial Blight Pathogenesis

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investigation of pathogenisity and genotypic diversity of xanthomonas oryzae pv. oryzae caused rice bacterial blight

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Promoter elements of rice susceptibility genes are bound and activated by specific TAL effectors from the bacterial blight pathogen, Xanthomonas oryzae pv. oryzae.

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Study of Biochemical and Molecular Changes of Iranian Rice Cultivars in Interaction with Bacterial Pathogen Xanthomonas oryzae pv. oryzae Causes Leaf Blight Disease

Rice bacterial blight caused by Xanthamonos oryzae pv. oryzae is one of the most destructive bacterial diseases of rice in some areas of rice cultivation in the world, especially in the tropics of Asia. The low efficiency of disease management methods, especially chemical methods, has led to more research on recognizing resistant cultivars and understanding resistance mechanisms through the stu...

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Xanthomonas oryzae pathovars: model pathogens of a model crop.

SUMMARY Xanthomonas oryzae pv. oryzae and Xanthomonas oryzae pv. oryzicola cause bacterial blight and bacterial leaf streak of rice (Oryza sativa), which constrain production of this staple crop in much of Asia and parts of Africa. Tremendous progress has been made in characterizing the diseases and breeding for resistance. X. oryzae pv. oryzae causes bacterial blight by invading the vascular t...

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The genome sequence of Xanthomonas oryzae pathovar oryzae KACC10331, the bacterial blight pathogen of rice

The nucleotide sequence was determined for the genome of Xanthomonas oryzae pathovar oryzae (Xoo) KACC10331, a bacterium that causes bacterial blight in rice (Oryza sativa L.). The genome is comprised of a single, 4 941 439 bp, circular chromosome that is G + C rich (63.7%). The genome includes 4637 open reading frames (ORFs) of which 3340 (72.0%) could be assigned putative function. Orthologs ...

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ژورنال

عنوان ژورنال: Indian Phytopathology

سال: 2017

ISSN: 2248-9800,0367-973X

DOI: 10.24838/ip.2017.v70.i3.72486