Dexras1, a Small GTPase, Is Required for Glutamate-NMDA Neurotoxicity

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Dexras1, a small GTPase, is required for glutamate-NMDA neurotoxicity.

Dexras1, a small G-protein localized predominantly to the brain, is transcriptionally upregulated by the synthetic glucocorticoid dexamethasone. It has close homology to the Ras subfamily but differs in that Dexras1 contains an extended 7 kDa C-terminal tail. Previous studies in our laboratory showed that NMDA receptor activation, via NO and Dexras1, physiologically stimulates DMT1, the major i...

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Dexras1 is a 30 kDa G protein in the Ras subfamily whose discovery was based on its pronounced inducibility by the glucocorticoid dexamethasone. It binds to neuronal nitric oxide synthase (nNOS) via the adaptor protein CAPON, eliciting S-nitrosylation and activation of Dexras1. We report that Dexras1 binds to the peripheral benzodiazepine receptor-associated protein (PAP7), a protein of unknown...

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Glutamate but not glycine agonist affinity for NMDA receptors is influenced by small cations.

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Mitochondrial dysfunction is a primary event in glutamate neurotoxicity.

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ژورنال

عنوان ژورنال: Journal of Neuroscience

سال: 2013

ISSN: 0270-6474,1529-2401

DOI: 10.1523/jneurosci.1497-12.2013