Characterization of Xanthomonas campestris pv. pennamericanum pv. nov., Causal Agent of Bacterial Leaf Streak of Pearl Millet

Genetic Diversity and Phylogenetic Study of Xanthomonas arboricola pv. juglandis the Causal Agent of Walnut Bacterial Blight Disease

Bacterial blight (Xanthomonas arboricola pv.juglandis) is one of the main diseases of walnut that reduce the yield in the central, western and northern regions of Iran. This disease was first reported form, Qazvin and Mazandaran, then widely reported from northern, central and western provinces. To identify the cause of the disease in different provinces, infected leaves collected from Alborze,...

Biological role of xanthomonadin pigments in Xanthomonas campestris pv. campestris.

Previous studies have indicated that the yellow pigments (xanthomonadins) produced by phytopathogenic Xanthomonas bacteria are unimportant during pathogenesis but may be important for protection against photobiological damage. We used a Xanthomonas campestris pv. campestris parent strain, single-site transposon insertion mutant strains, and chromosomally restored mutant strains to define the bi...

Genome Sequence of Xanthomonas campestris pv. campestris Strain Xca5

An annotated high-quality draft genome sequence for Xanthomonas campestris pv. campestris race 1 strain Xca5 (originally described as X. campestris pv. armoraciae), the causal agent of black rot on Brassicaceae plants, has been determined. This genome sequence is a valuable resource for comparative genomics within the campestris pathovar.

Xanthomonas campestris pv. campestris race 1 is the main causal agent of black rot of Brassicas in Southern Mozambique

Bactericidal activity of glycinecin A, a bacteriocin derived from Xanthomonas campestris pv. glycines, on phytopathogenic Xanthomonas campestris pv. vesicatoria cells.

The ability of glycinecin A, a bacteriocin derived from Xanthomonas campestris pv. glycines 8ra, to kill closely related bacteria has been demonstrated previously by our group. In the present study, we aimed at determining the glycinecin A-induced cause of death. Treatment with glycinecin A caused slow dissipation of membrane potential and rapid depletion of the pH gradient. Glycinecin A treatm...