Characterization of a cDNA Encoding CuZn-Superoxide Dismutase from the Liverwort Marchantia paleacea var. diptera
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چکیده
منابع مشابه
Light-dependent expression of protochlorophyllide oxidoreductase gene in the liverwort, Marchantia paleacea var. diptera.
A cDNA encoding the NADPH:protochlorophyllide oxidoreductase (EC 1.6.99.1) was isolated from suspension-cultured cells of the liverwort, Marchantia paleacea var. diptera. In contrast to the situation in most higher plants, the liverwort gene was expressed in a light-dependent manner.
متن کاملPhotosynthetic electron transport regulates the stability of the transcript for the protochlorophyllide oxidoreductase gene in the liverwort, Marchantia paleacea var. diptera.
The transfer of Marchantia paleacea var. diptera cells to darkness caused a reversible repression in the accumulation of transcript for a gene, por, encoding the NADPH: protochlorophyllide oxidoreductase (EC 1.3.1.33). The photosynthetic inhibitor DCMU and DBMIB repressed the accumulation in light. In the presence of transcription inhibitor cordycepin, not only incubation in the dark but also a...
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_, We have used female and male transgenic (Tg) mice that carry the complete sequence of the human copper-zinc (CuZn) , superoxide dismutase (SOD) gene in order to assess the lethal effects of methylenedioxyamphetamine (MDA) and methylene,_ im dioxymethamphetamine (MDMA). In contrast to non-Tg mice, both heterozygous and homozygous SOD-Tg mice showed resistance to the lethal effects of both dru...
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Suspension cultured cells of the liverwort Marchantia paleacea var. diptera (MpK) were investigated to clarify the relationship between morphogenesis and the mode of utilization of nitrogen sources in different media. MpK cells grew vigorously in a Murashige and Skoog's liquid medium with 4% glucose in the light condition. Thallus regeneration from cultured cells occurred in culture medium with...
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Mutations in CuZn-superoxide dismutase (CuZn-SOD) have been linked to familial amyotrophic lateral sclerosis (ALS), and motor neurone death is caused by the gain of a toxic property of the mutant protein. Here we determined amounts, activity and molecular forms of CuZn-SOD in CSF from ALS patients carrying the D90A and other CuZn-SOD mutations and patients without such mutations. There were no ...
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ژورنال
عنوان ژورنال: Plant and Cell Physiology
سال: 1998
ISSN: 0032-0781,1471-9053
DOI: 10.1093/oxfordjournals.pcp.a029362