Bithalamic Lesions

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چکیده

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منابع مشابه

Bithalamic dysfunction in Wernicke's encephalopathy

We describe a 65-year-old, nonalcoholic, right-handed female with multiple vascular risk factors who developed transient visuospatial hemineglect and global aphasia after presenting with the classic triad of Wernicke's encephalopathy (mental status changes, nystagmus/ophthalmoplegia, and ataxia). A brain MRI showed no evidence of acute infarction, but demonstrated signal change in the medial th...

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Akinetic mutism with bithalamic infarction. Neurophysiological correlates.

A 42-year-old man presented with akinetic mutism, caused by bilateral thalamic infarction, resulting in a temporary amnesic syndrome and, finally, selective downgaze palsy. Electroencephalographic recording in the initial phase showed generalized spike- and-wave discharges, similar to those found in animal studies after lesion or stimulation of specific thalamic nuclei. Analysis of magnetic res...

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Venous hypertension associated with a posterior fossa dural arteriovenous fistula: another cause of bithalamic lesions on MR images.

We report a case of a posterior fossa arteriovenous fistula (AVF) with bithalamic hyperintensity of MR images. The thalamic abnormality improved after surgery, suggesting reversible venous hypertension as the pathogenesis of the finding, as opposed to infarction. This manifestation of a posterior fossa AVF should be considered in the differential diagnosis of bilateral thalamic disease.

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Bithalamic compromise in acute disseminated encephalomyelitis following H1N1 influenza vaccine.

Received 4 July 2010. Received in final form 18 January 2011. Accepted 25 January 2011. A 5-year-old boy was admitted with history of fever, nausea, vomiting and somnolence which had started two days after H1N1 influenza vaccination. Neurological examination showed mild gait ataxia, vertical ophthalmoparesis and brisk deep tendon reflexes. Brain MRI revealed right frontal and bilateral thalamic...

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Loss of psychic self-activation after paramedian bithalamic infarction.

BACKGROUND AND PURPOSE Loss of psychic self-activation has been described after bilateral lesions to the globus pallidus, striatum, and white matter of the frontal lobes, but it is a very rare sign of bithalamic lesions. The exact functional-anatomic mechanism underlying loss of psychic self-activation following bithalamic lesions remains to be elucidated. CASE DESCRIPTION We present clinical...

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ژورنال

عنوان ژورنال: Stroke

سال: 2020

ISSN: 0039-2499,1524-4628

DOI: 10.1161/strokeaha.120.030576