Bacterial Black Stem Rot on Angelica acutiloba Caused by Xanthomonas campestris
نویسندگان
چکیده
منابع مشابه
Characterization of the GntR family regulator HpaR1 of the crucifer black rot pathogen Xanthomonas campestris pathovar campestris
The GntR family transcription regulator HpaR1 identified from Xanthomonas campestris pv. campestris has been previously shown to positively regulate the genes responsible for hypersensitive reaction and pathogenicity and to autorepress its own expression. Here, we demonstrated that HpaR1 is a global regulator that positively regulates diverse biological processes, including xanthan polysacchari...
متن کاملXanthomonas campestris pv. campestris race 1 is the main causal agent of black rot of Brassicas in Southern Mozambique
متن کامل
Xanthomonas campestris pv. campestris (cause of black rot of crucifers) in the genomic era is still a worldwide threat to brassica crops.
BACKGROUND Xanthomonas campestris pv. campestris (Xcc) (Pammel) Dowson is a Gram-negative bacterium that causes black rot, the most important disease of vegetable brassica crops worldwide. Intensive molecular investigation of Xcc is gaining momentum and several whole genome sequences are available. TAXONOMY Bacteria; Phylum Proteobacteria; Class Gammaproteobacteria; Order Xanthomonadales; Fam...
متن کاملEvaluation of the potential of the flea beetle Phyllotreta cruciferae to transmit Xanthomonas campestris pv. campestris, causal agent of black rot of crucifers
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Angelica acutiloba Kitagawa Extract Attenuates DSS-Induced Murine Colitis
We examined the protective effects of Angelica acutiloba Kitagawa (AAK) extract on a murine model of acute experimental colitis. Colitis was induced by 4% dextran sulfate sodium (DSS) in the drinking water of male C57BL/6 mice, for 7 consecutive days. Oral administration of AAK extract (500 mg/kg/day) significantly alleviated DSS-induced symptoms such as anorexia, weight loss, events of diarrhe...
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ژورنال
عنوان ژورنال: The Plant Pathology Journal
سال: 2002
ISSN: 1598-2254
DOI: 10.5423/ppj.2002.18.1.054