Acute hyperammonaemic encephalopathy

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منابع مشابه

Acute hyperammonaemic encephalopathy

Peng Soon Ng1,*, Choie Cheio Tchoyoson Lim2 and Nigel CK Tan1 Department of Neurology, National Neuroscience Institute, Singapore, Singapore and Department of Neuroradiology, National Neuroscience Institute, Singapore, Singapore *Correspondence address. Department of Neurology, National Neuroscience Institute, 11 Jalan Tan Tock Seng, Singapore 308433. Tel: þ65-63577171; Fax: þ65-63577137; E-mai...

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Valproic acid induced hyperammonaemic encephalopathy.

OBJECTIVE To observe clinical and laboratory features of valproic acid-induced hyperammonaemic encephalopathy in patients taking valproic acid. METHODS Observational study was conducted at the Neurology Department, Dow University of Health Sciences, Civil Hospital, Karachi, from February 26, 2010 to March 20, 2011. Ten patients on valproic acid therapy of any age group with idiopathic or seco...

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Hyperammonaemic encephalopathy after a subureteric injection for vesicoureteric reflux.

A 6 year old boy developed hyperammonaemic encephalopathy following a subureteric injection for treatment of vesicoureteric reflux. The hyperammonaemia may be explained by a postoperative urinary tract infection with a urea splitting organism, leading to raised urine ammonia that was absorbed easily across a dilated urinary tract. Agitation and alteration in consciousness level following a urol...

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Non-hyperammonaemic valproate encephalopathy after 20 years of treatment

Sodium valproate is a commonly used antiseizure drug with broad indications for different seizuretypes and epilepsy syndromes. Well-recognised side effects include weight gain, tremor, dizziness, and unsteadiness. Non-hyperammonaemic parkinsonism, with or without cognitive impairment, is a rare adverse effect of sodium valproate. We present the case of a sixty year-old lady with a generalized s...

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A case of hyperammonaemic encephalopathy due to valproic acid.

A patient with valproic acid induced hyperammonaemic encephalopathy is presented. During chronic treatment with valproic acid this patient developed a potentially life-threatening encephalopathy without signs of liver failure. After discontinuing the valproic acid the patient recovered completely. In the case of a patient presenting with hyperammonaemic encephalopathy, the possibility of the us...

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ژورنال

عنوان ژورنال: Oxford Medical Case Reports

سال: 2014

ISSN: 2053-8855

DOI: 10.1093/omcr/omu027