نتایج جستجو برای: bethlem myopathy

تعداد نتایج: 12325  

Journal: :Journal of the Formosan Medical Association = Taiwan yi zhi 2001
L M Lien C C Yang W H Chen H C Chiu

We report three cases of Bethlem myopathy from three consecutive generations of a Taiwanese family, including one woman aged 70, one man aged 40, and a boy aged 8. The clinical features of the patients included autosomal dominant inheritance, childhood or adolescent onset, mainly proximal and extensor involvement, early diffuse joint contractures, and absence of cardiac involvement. These featu...

Journal: :Pediatric Neurology Briefs 1999

2015
Hyung Jun Park Young-Chul Choi Seung Min Kim Se Hoon Kim Young Bin Hong Bo Ram Yoon Ki Wha Chung Byung-Ok Choi

BACKGROUND We describe herein the application of whole exome sequencing (WES) for the molecular genetic diagnosis of a large Korean family with dominantly inherited myopathy. CASE REPORT The affected individuals presented with slowly progressive proximal weakness and ankle contracture. They were initially diagnosed with limb-girdle muscular dystrophy (LGMD) based on clinical and pathologic fe...

2017

The collagen type VI-related disorders are nowadays considered to be a continuum of overlapping phenotypes with Bethlem myopathy at the mild end and Ullrich congenital muscular dystrophy (UCMD) at the severe end. In between these phenotypes there are collagen type VI-related limb-girdle muscular dystrophy and myosclerosis myopathy. Most cases of Bethlem myopathy have autosomal dominant inherita...

Journal: :Human molecular genetics 1996
M C Speer R Tandan P N Rao T Fries J M Stajich P A Bolhuis G J Jöbsis J M Vance K D Viles K Sheffield C James S G Kahler M Pettenati J R Gilbert P H Denton L H Yamaoka M A Pericak-Vance

The Bethlem myopathy, a childhood onset autosomal dominant myopathy with joint contractures, has recently been localized to 21q in a series of Dutch families and the alpha 1 and alpha 2 subunits of type VI collagen (COL6A1 and COL6A2) have been postulated as candidate genes. We investigate a large family of French Canadian descent (family 1489) in which the Bethlem myopathy is segregating. Fami...

2015

Synonyms Spectrum of phenotypes: Mild: Bethlem myopathy/ benign congenital muscular dystrophy Intermediate: Limb-girdle muscular dystrophy; myosclerosis myopathy Severe: Ullrich myopathy/ congenital atonic sclerotic muscular dystrophy First described by Ullrich in 1930 and Bethlem in 1976 respectively [1]. Caused by mutations in any of the 3 genes which code for collagen type VI synthesis, COL6...

Journal: :Brain : a journal of neurology 1999
G J Jöbsis J M Boers P G Barth M de Visser

Bethlem myopathy is an early-onset benign autosomal dominant myopathy with contractures caused by mutations in collagen type VI genes. It has been reported that onset occurs in early childhood. We investigated the natural course of Bethlem myopathy in five previously published kindreds and two novel pedigrees, with particular attention to the mode of onset in 23 children and the progression of ...

Journal: :Archives of neurology 2006
Anneke J van der Kooi Willem G de Voogt Enrico Bertini Luciano Merlini F Beril Talim Rabah Ben Yaou Andoni Urtziberea Marianne de Visser

BACKGROUND Bethlem myopathy is considered a relatively mild neuromuscular disorder without significant cardiac and respiratory involvement. OBJECTIVE To investigate cardiac and respiratory involvement in Bethlem myopathy. DESIGN Cross-sectional study. SETTING University hospitals. Patients Fifty patients with Bethlem myopathy from 26 families. INTERVENTIONS Cardiac examinations, includi...

2014
Alessandra Zulian Francesca Tagliavini Erika Rizzo Camilla Pellegrini Francesca Sardone Nicoletta Zini Nadir Mario Maraldi Spartaco Santi Cesare Faldini Luciano Merlini Valeria Petronilli Paolo Bernardi Patrizia Sabatelli

Ullrich congenital muscular dystrophy and Bethlem myopathy are caused by mutations in collagen VI (ColVI) genes, which encode an extracellular matrix protein; yet, mitochondria play a major role in disease pathogenesis through a short circuit caused by inappropriate opening of the permeability transition pore, a high-conductance channel, which causes a shortage in ATP production. We find that m...

Journal: :Neurology 2012
James Collins A Reghan Foley Volker Straub Carsten G Bönnemann

A 32-year-old woman and a 50-year-old man with clinically typical Bethlem myopathy developed seemingly spontaneous keloids on their shoulder region (figure). The patients did not recall any significant trauma to the skin of this region. Bethlem myopathy (MIM #158810) is caused by dominant and recessive mutations in the collagen VI genes: COL6A1, COL6A2, and COL6A3. Skin manifestations include h...

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