نتایج جستجو برای: SLC47A1

تعداد نتایج: 90  

2015
Tomohiro Mizuno Waichi Sato Kazuhiro Ishikawa Yuki Terao Kazuo Takahashi Yukihiro Noda Yukio Yuzawa Tadashi Nagamatsu

BACKGROUND/AIM To elucidate the mechanism responsible for developing acute kidney injury in patients with diabetes mellitus, we also evaluated the issue of whether advanced glycation endproducts (AGEs) influence the expressions of multi antimicrobial extrusion protein (MATE1/SLC47A1) in tubular cells. MATERIALS AND METHODS To detect changing expression of MATE1/SLC47A1 in dose- and time-depen...

Journal: :Molecular pharmacology 2018
Toshihiro Tanaka Takeshi Hirota Ichiro Ieiri

Multidrug and toxin extrusion protein 1 (MATE1), which is encoded by solute carrier 47A1 (SLC47A1), mediates the excretion of organic cations into bile and urine. Some genetic variants in human MATE1 altered its transport function in in vitro experiments; however, differences in the pharmacokinetics of substrate drugs cannot be explained by genetic variations in humans. In this study, we invest...

2017
Gerard Marshall Raj Jayanthi Mathaiyan Mukta Wyawahare Katiboina Srinivasa Rao Rekha Priyadarshini

Introduction: Drug transporters are key determinants of pharmacokinetic and pharmacodynamic profiles of certain drugs. SLC47A1 (MATE1) and SLC47A2 (MATE2) are major efflux transporters involved in the hepatic and renal excretion of many cationic drugs including metformin. Our study was proposed to determine the normative frequencies of the single nucleotide polymorphisms (SNPs) rs2289669 and rs...

Journal: :Diabetes 2009
Matthijs L. Becker Loes E. Visser Ron H.N. van Schaik Albert Hofman André G. Uitterlinden Bruno H.Ch. Stricker

OBJECTIVE Metformin, an oral glucose-lowering drug, is taken up in hepatocytes by the organic cation transporter (OCT) 1 and in renal epithelium by OCT2. In these cells, the multidrug and toxin extrusion (MATE) 1 protein, encoded by the SLC47A1 gene, is responsible for the excretion of metformin into the bile and urine, respectively. We studied the effect of single nucleotide polymorphisms (SNP...

2014
Antonio Brunetti

variation in the response to metformin has been observed in individuals with genetic variations in the genes coding for MATE1 (SLC47A1) and MATE2 (SLC47A2), which are involved in the extrusion of metformin through the urine. Whereas a greater response to metformin, with reduced levels of HbA1c, was found in association with the rs2252281 variant in the SLC47A1 gene, a reduced response to metfor...

Journal: :Physiology 2010
Catriona M H Anderson David T Thwaites

The physiological role of mammalian solute carrier (SLC) proteins is to mediate transmembrane movement of electrolytes, nutrients, micronutrients, vitamins, and endogenous metabolites from one cellular compartment to another. Many transporters in the small intestine, kidney, and solid tumors are H(+)-coupled, driven by local H(+)-electrochemical gradients, and transport numerous drugs. These tr...

2012
Cristian Pattaro Anna Köttgen Alexander Teumer Maija Garnaas Carsten A. Böger Christian Fuchsberger Matthias Olden Ming-Huei Chen Adrienne Tin Daniel Taliun Man Li Xiaoyi Gao Mathias Gorski Qiong Yang Claudia Hundertmark Meredith C. Foster Conall M. O'Seaghdha Nicole Glazer Aaron Isaacs Ching-Ti Liu Albert V. Smith Jeffrey R. O'Connell Maksim Struchalin Toshiko Tanaka Guo Li Andrew D. Johnson Hinco J. Gierman Mary Feitosa Shih-Jen Hwang Elizabeth J. Atkinson Kurt Lohman Marilyn C. Cornelis Åsa Johansson Anke Tönjes Abbas Dehghan Vincent Chouraki Elizabeth G. Holliday Rossella Sorice Zoltan Kutalik Terho Lehtimäki Tõnu Esko Harshal Deshmukh Sheila Ulivi Audrey Y. Chu Federico Murgia Stella Trompet Medea Imboden Barbara Kollerits Giorgio Pistis Tamara B. Harris Lenore J. Launer Thor Aspelund Gudny Eiriksdottir Braxton D. Mitchell Eric Boerwinkle Helena Schmidt Margherita Cavalieri Madhumathi Rao Frank B. Hu Ayse Demirkan Ben A. Oostra Mariza de Andrade Stephen T. Turner Jingzhong Ding Jeanette S. Andrews Barry I. Freedman Wolfgang Koenig Thomas Illig Angela Döring H.-Erich Wichmann Ivana Kolcic Tatijana Zemunik Mladen Boban Cosetta Minelli Heather E. Wheeler Wilmar Igl Ghazal Zaboli Sarah H. Wild Alan F. Wright Harry Campbell David Ellinghaus Ute Nöthlings Gunnar Jacobs Reiner Biffar Karlhans Endlich Florian Ernst Georg Homuth Heyo K. Kroemer Matthias Nauck Sylvia Stracke Uwe Völker Henry Völzke Peter Kovacs Michael Stumvoll Reedik Mägi Albert Hofman Andre G. Uitterlinden Fernando Rivadeneira Yurii S. Aulchenko Ozren Polasek Nick Hastie Veronique Vitart Catherine Helmer Jie Jin Wang Daniela Ruggiero Sven Bergmann Mika Kähönen Jorma Viikari Tiit Nikopensius Michael Province Shamika Ketkar Helen Colhoun Alex Doney Antonietta Robino Franco Giulianini Bernhard K. Krämer Laura Portas Ian Ford Brendan M. Buckley Martin Adam Gian-Andri Thun Bernhard Paulweber Margot Haun Cinzia Sala Marie Metzger Paul Mitchell Marina Ciullo Stuart K. Kim Peter Vollenweider Olli Raitakari Andres Metspalu Colin Palmer Paolo Gasparini Mario Pirastu J. Wouter Jukema Nicole M. Probst-Hensch Florian Kronenberg Daniela Toniolo Vilmundur Gudnason Alan R. Shuldiner Josef Coresh Reinhold Schmidt Luigi Ferrucci David S. Siscovick Cornelia M. van Duijn Ingrid Borecki Sharon L. R. Kardia Yongmei Liu Gary C. Curhan Igor Rudan Ulf Gyllensten James F. Wilson Andre Franke Peter P. Pramstaller Rainer Rettig Inga Prokopenko Jacqueline C. M. Witteman Caroline Hayward Paul Ridker Afshin Parsa Murielle Bochud Iris M. Heid Wolfram Goessling Daniel I. Chasman W. H. Linda Kao Caroline S. Fox

Chronic kidney disease (CKD) is an important public health problem with a genetic component. We performed genome-wide association studies in up to 130,600 European ancestry participants overall, and stratified for key CKD risk factors. We uncovered 6 new loci in association with estimated glomerular filtration rate (eGFR), the primary clinical measure of CKD, in or near MPPED2, DDX1, SLC47A1, C...

2016
Kazuma Fujita Takenori Niioka Masatomo Miura

Background: The individual and collective contributions of genetic polymorphisms in drug transporter genes in human renal proximal tubules to cisplatin-induced nephrotoxicity are still unclear. Methods: In this study, we investigated the effects of polymorphisms in SLC22A2 (808G>T), SLC31A1 (rs10981694A>C, rs12686377G>T, rs7851395A>G), SLC47A1 (rs2289669G>A), ABCB1 (1236C>A, 2677G>T/A, 3435C>T)...

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