نتایج جستجو برای: SLC29A3 gene mutation

تعداد نتایج: 1284717  

Journal: :Pediatrics 2013
Isabelle Melki Karen Lambot Laurence Jonard Vincent Couloigner Pierre Quartier Bénédicte Neven Brigitte Bader-Meunier

Germline mutations in the SLC29A3 gene result in a range of recessive, clinically related syndromes: H syndrome, pigmented hypertrichosis with insulin-dependent diabetes mellitus syndrome, Faisalabad histiocytosis, and sinus histiocytosis with massive lymphadenopathy. The main symptoms of these diseases are hyperpigmentation with hypertrichosis, sensorineural deafness, diabetes, short stature, ...

Journal: :Chinese medical journal 2015
Jia-Wei Liu Nuo Si Lian-Qing Wang Ti Shen Xue-Jun Zeng Xue Zhang Dong-Lai Ma

BACKGROUND H syndrome (OMIM 612391) is a recently described autosomal recessive genodermatosis characterized by indurated hyperpigmented and hypertrichotic skin, as well as other systemic manifestations. Most of the cases occurred in the Middle East areas or nearby countries such as Spain or India. The syndrome is caused by mutations in solute carrier family 29, member 3 (SLC29A3), the gene enc...

2013
Laurence Jonard Vincent Couloigner

Germline mutations in the SLC29A3 gene result in a range of recessive, clinically related syndromes: H syndrome, pigmented hypertrichosis with insulin-dependent diabetes mellitus syndrome, Faisalabad histiocytosis, and sinus histiocytosis with massive lymphadenopathy. The main symptoms of these diseases are hyperpigmentation with hypertrichosis, sensorineural deafness, diabetes, short stature, ...

2010
Neil V. Morgan Mark R. Morris Hakan Cangul Diane Gleeson Anna Straatman-Iwanowska Nicholas Davies Stephen Keenan Shanaz Pasha Fatimah Rahman Dean Gentle Maaike P. G. Vreeswijk Peter Devilee Margaret A. Knowles Serdar Ceylaner Richard C. Trembath Carlos Dalence Erol Kismet Vedat Köseoğlu Hans-Christoph Rossbach Paul Gissen David Tannahill Eamonn R. Maher

The histiocytoses are a heterogeneous group of disorders characterised by an excessive number of histiocytes. In most cases the pathophysiology is unclear and treatment is nonspecific. Faisalabad histiocytosis (FHC) (MIM 602782) has been classed as an autosomal recessively inherited form of histiocytosis with similarities to Rosai-Dorfman disease (RDD) (also known as sinus histiocytosis with ma...

Journal: :Indian Journal of Paediatric Dermatology 2020

Journal: :Human molecular genetics 2009
Simon T Cliffe Jamie M Kramer Khalid Hussain Joris H Robben Eiko K de Jong Arjan P de Brouwer Esther Nibbeling Erik-Jan Kamsteeg Melanie Wong Julie Prendiville Chela James Raja Padidela Charlie Becknell Hans van Bokhoven Peter M T Deen Raoul C M Hennekam Robert Lindeman Annette Schenck Tony Roscioli Michael F Buckley

Pigmented hypertrichotic dermatosis with insulin-dependent diabetes (PHID) syndrome is a recently described autosomal recessive disorder associated with predominantly antibody negative, insulin-dependent diabetes mellitus. In order to identify the genetic basis of PHID and study its relationship with glucose metabolism, we performed homozygosity mapping in five unrelated families followed by ca...

Background  H syndrome is an autosomal recessive genodermatosis with a low prevalence which is caused by a mutation in SLC29A3 gene. This disorder is characterized by sclerotic, hyperpigmented, hypertrichotic cutaneous plaques with systemic involvement including: hepatosplenomegaly, heart anomalies, hearing loss, hypogonadism, low height, and hyperglycemia. Case Presentation  Here we have pres...

Journal: :Human molecular genetics 2012
Philippe M Campeau James T Lu Gautam Sule Ming-Ming Jiang Yangjin Bae Simran Madan Wolfgang Högler Nicholas J Shaw Steven Mumm Richard A Gibbs Michael P Whyte Brendan H Lee

Dysosteosclerosis (DSS) is the form of osteopetrosis distinguished by the presence of skin findings such as red-violet macular atrophy, platyspondyly and metaphyseal osteosclerosis with relative radiolucency of widened diaphyses. At the histopathological level, there is a paucity of osteoclasts when the disease presents. In two patients with DSS, we identified homozygous or compound heterozygou...

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