نتایج جستجو برای: PKHD1

تعداد نتایج: 158  

2017
Su Lin Cheng He Min-Fang Wang Yin-Lian Wu Jian Lin Yi Liu Yue-Yong Zhu

Intrahepatic cholangiocarcinoma (ICC), a type of cholangiocarcinoma, is characterized by insidious onset and lack of typical clinical symptoms at early onset, and the lack of effective treatments results in a poor prognosis. Identification of novel biomarkers and treatment targets is therefore of great significance to improve the survival for ICC patients. Polycystic kidney and hepatic disease ...

Journal: :American journal of physiology. Renal physiology 2014
Scott S Williams Patricia Cobo-Stark Sachin Hajarnis Karam Aboudehen Xinli Shao James A Richardson Vishal Patel Peter Igarashi

Autosomal recessive polycystic kidney disease, an inherited disorder characterized by the formation of cysts in renal collecting ducts and biliary dysgenesis, is caused by mutations of the polycystic kidney and hepatic disease 1 (PKHD1) gene. Expression of PKHD1 is tissue specific and developmentally regulated. Here, we show that a 2.0-kb genomic fragment containing the proximal promoter of mou...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2004
Ming-Zhi Zhang Weiyi Mai Cunxi Li Sae-youll Cho Chuanming Hao Gilbert Moeckel Runxiang Zhao Ingyu Kim Jikui Wang Huaqi Xiong Hong Wang Yasunori Sato Yizhong Wu Yasuni Nakanuma Marusia Lilova York Pei Raymond C Harris Song Li Robert J Coffey Le Sun Dianqing Wu Xing-Zhen Chen Matthew D Breyer Zhizhuang Joe Zhao James A McKanna Guanqing Wu

Mutations of the polycystic kidney and hepatic disease 1 (PKHD1) gene have been shown to cause autosomal recessive polycystic kidney disease (ARPKD), but the cellular functions of the gene product (PKHD1) remain uncharacterized. To illuminate its properties, the spatial and temporal expression patterns of PKHD1 were determined in mouse, rat, and human tissues by using polyclonal Abs and mAbs re...

2014
Michaela Drögemüller Vidhya Jagannathan Monika M. Welle Claudia Graubner Reto Straub Vinzenz Gerber Dominik Burger Heidi Signer-Hasler Pierre-André Poncet Stéphane Klopfenstein Ruedi von Niederhäusern Jens Tetens Georg Thaller Stefan Rieder Cord Drögemüller Tosso Leeb William Barendse

Congenital hepatic fibrosis has been described as a lethal disease with monogenic autosomal recessive inheritance in the Swiss Franches-Montagnes horse breed. We performed a genome-wide association study with 5 cases and 12 controls and detected an association on chromosome 20. Subsequent homozygosity mapping defined a critical interval of 952 kb harboring 10 annotated genes and loci including ...

2015
K. J. Kelly Jizhong Zhang Ling Han Malgorzata Kamocka Caroline Miller Vincent H. Gattone Jesus H. Dominguez Benedetta Bussolati

Autosomal recessive polycystic kidney disease is a truly catastrophic monogenetic disease, causing death and end stage renal disease in neonates and children. Using PCK female rats, an orthologous model of autosomal recessive polycystic kidney disease harboring mutant Pkhd1, we tested the hypothesis that intravenous renal cell transplantation with normal Sprague Dawley male kidney cells would i...

2014
Xiwei Hao Shiguo Liu Qian Dong Hong Zhang Jing Zhao Lin Su

BACKGROUND Mutations in PKHD1 cause autosomal recessive Caroli disease, which is a rare congenital disorder involving cystic dilatation of the intrahepatic bile ducts. However, the mutational spectrum of PKHD1 and the phenotype-genotype correlations have not yet been fully established. METHODS Whole exome sequencing (WES) was performed on one twin sample with Caroli disease from a Chinese fam...

Journal: :World journal of pediatrics : WJP 2013
Xi-Hui Zhou Zhi-Yan Hui Yuan Li

BACKGROUND Autosomal recessive polycystic kidney disease (ARPKD) is one of the most common hereditary nephropathies in childhood. We report a neonate with ARPKD presenting with oligohydramnios, enlargement and increased echogenicity of both kidneys shown by antenatal sonograms after a 29-week gestation and died within the first few hours of life. METHODS The neonate was investigated pathologi...

Journal: :Human molecular genetics 2007
Miguel A Garcia-Gonzalez Luis F Menezes Klaus B Piontek Junya Kaimori David L Huso Terry Watnick Luiz F Onuchic Lisa M Guay-Woodford Gregory G Germino

Polycystic kidney disease (PKD) describes a heterogeneous collection of disorders that differ significantly with respect to their etiology and clinical presentation. They share, however, abnormal tubular morphology as a common feature, leading to the hypothesis that their respective gene products may function cooperatively in a common pathway to maintain tubular integrity. To study the pathobio...

Journal: :Journal of the American Society of Nephrology : JASN 2008
Ingyu Kim Yulong Fu Kwokyin Hui Gilbert Moeckel Weiyi Mai Cunxi Li Dan Liang Ping Zhao Jie Ma Xing-Zhen Chen Alfred L George Robert J Coffey Zhong-Ping Feng Guanqing Wu

Autosomal recessive polycystic kidney disease is caused by mutations in PKHD1, which encodes the membrane-associated receptor-like protein fibrocystin/polyductin (FPC). FPC associates with the primary cilia of epithelial cells and co-localizes with the Pkd2 gene product polycystin-2 (PC2), suggesting that these two proteins may function in a common molecular pathway. For investigation of this, ...

Journal: :Journal of medical genetics 2005
C Bergmann F Küpper C P Schmitt U Vester T J Neuhaus J Senderek K Zerres

BACKGROUND Autosomal recessive polycystic kidney disease (ARPKD) is caused by mutations in the PKHD1 (polycystic kidney and hepatic disease 1) gene on chromosome 6p12, a large gene spanning 470 kb of genomic DNA. So far, only micromutations in the 66 exons encoding the longest open reading frame (ORF) have been described, and account for about 80% of mutations. OBJECTIVE To test the hypothesi...

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