نتایج جستجو برای: سولفیدسدیم (Na2S)

تعداد نتایج: 203  

ژورنال: :نشریه علمی-پژوهشی مهندسی معدن 2015
محمود عبدالهی بیژن طاهری سید ضیاالدین شفایی تنکابنی سهیلا جوادیان فرزانه

.در این تحقیق، رفتار شناورسازی کالکوپیریت بصورت تک کانی و ترکیبی (کنسانتره کالکوپیریت) در حضور مقادیر مختلف سولفیدسدیم در سه سطح ph (7، 9 و 12) تحت کنترل پتانسیل الکتروشیمیایی پالپ مورد بررسی قرار گرفت. از روش اسپکتروسکوپی فتوالکترون اشعه ایکس (xps) بعنوان یک روش توانمند آنالیز سطح برای شناسایی ترکیبات سطحی هیدروفیل و هیدروفوب به منظور تحلیل مکانیزم شناورسازی و بازداشت کالکوپیریت در شرایط مختلف...

2014
Jonathan P Lambert Chad K Nicholson Hena Amin Sana Amin John W Calvert

BACKGROUND Coronary artery disease remains the principal cause of death in patients with diabetes mellitus. Diabetic mice display exacerbated injury following myocardial ischemia-reperfusion (MI/R) and are resistant to most therapeutic interventions. We have reported that sodium sulfide (Na2S) therapy confers cardioprotection during MI/R in non-diabetic mice. Here we tested the hypothesis that ...

2017
Shin Koike Shoichi Nishimoto Yuki Ogasawara

Many physiological functions of hydrogen sulfide (H2S) have been reported in mammalian cells over the last 20 years. These physiological effects have been ascertained through in vitro treatment of cells with Na2S or NaHS, both of which are precursors of H2S. Since H2S exists as HS- in a neutral solution, a disulfide compound such as cystine could react with HS- in culture medium as well as in t...

Journal: :Circulation. Heart failure 2016
Yuuki Shimizu Chad K Nicholson Jonathan P Lambert Larry A Barr Nicholas Kuek David Herszenhaut Lin Tan Toyoaki Murohara Jason M Hansen Ahsan Husain Nawazish Naqvi John W Calvert

BACKGROUND Therapeutic strategies aimed at increasing hydrogen sulfide (H2S) levels exert cytoprotective effects in various models of cardiovascular injury. However, the underlying mechanism(s) responsible for this protection remain to be fully elucidated. Nuclear factor E2-related factor 2 (Nrf2) is a cellular target of H2S and facilitator of H2S-mediated cardioprotection after acute myocardia...

Journal: :Circulation. Cardiovascular genetics 2014
Stefano Toldo Anindita Das Eleonora Mezzaroma Vinh Q Chau Carlo Marchetti David Durrant Arun Samidurai Benjamin W Van Tassell Chang Yin Ramzi A Ockaili Navin Vigneshwar Nitai D Mukhopadhyay Rakesh C Kukreja Antonio Abbate Fadi N Salloum

BACKGROUND Maintaining physiological levels of hydrogen sulfide during ischemia is necessary to limit injury to the heart. Because of the anti-inflammatory effects of hydrogen sulfide, we proposed that the hydrogen sulfide donor, sodium sulfide (Na2S), would attenuate myocardial injury through upregulation of protective microRNA-21 (miR-21) and suppression of the inflammasome, a macromolecular ...

Journal: :American journal of physiology. Heart and circulatory physiology 2015
Ryan C Jupiter Daniel Yoo Edward A Pankey Vishwaradh V G Reddy Justin A Edward David J Polhemus Taylor C Peak Prasad Katakam Philip J Kadowitz

Hydrogen sulfide (H2S) is a biologically active endogenous gasotransmitter formed in penile tissue that has been shown to relax isolated cavernosal smooth muscle. In the present study, erectile responses to the H2S donors sodium sulfide (Na2S) and sodium hydrosulfide (NaHS) were investigated in the anesthetized rat. Intracavernosal injections of Na2S in doses of 0.03-1 mg/kg increased intracave...

2013
BO WANG MEI LI XIAOGUANG LI XUAN FANG XIAN GAO YONGGANG ZOU PENG LU GUOJUN LIU XIAOHUI MA

In this paper, (NH4)2S and Na2S were used as passivating agent for the sulphuration treatment of GaSb. Although the oxide layer can be removed by both (NH4)2S and Na2S, the etching rate of Na2S was faster than (NH4)2S, which caused the high RMS. After passivation treatment, PL instensity of GaSb increased, however, if the passivation time was too long, PL instensity would decrease. At low tempe...

Journal: :American journal of physiology. Regulatory, integrative and comparative physiology 2015
Alisa M Agné Jan-Peter Baldin Audra R Benjamin Maria C Orogo-Wenn Lukas Wichmann Kenneth R Olson Dafydd V Walters Mike Althaus

In pulmonary epithelia, β-adrenergic agonists regulate the membrane abundance of the epithelial sodium channel (ENaC) and, thereby, control the rate of transepithelial electrolyte absorption. This is a crucial regulatory mechanism for lung liquid clearance at birth and thereafter. This study investigated the influence of the gaseous signaling molecule hydrogen sulfide (H2S) on β-adrenergic agon...

ژورنال: مهندسی معدن 2015

.در این تحقیق، رفتار شناورسازی کالکوپیریت بصورت تک‌کانی و ترکیبی (کنسانتره کالکوپیریت) در حضور مقادیر مختلف سولفیدسدیم در سه سطح pH (7، 9 و 12) تحت کنترل پتانسیل الکتروشیمیایی پالپ مورد بررسی قرار گرفت. از روش اسپکتروسکوپی فتوالکترون اشعه ایکس (XPS) بعنوان یک روش توانمند آنالیز سطح برای شناسایی ترکیبات سطحی هیدروفیل و هیدروفوب به منظور تحلیل مکانیزم شناورسازی و بازداشت کالکوپیریت در شرایط مختلف...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2013
Chad K Nicholson Jonathan P Lambert Jeffery D Molkentin Junichi Sadoshima John W Calvert

OBJECTIVE The aim of this study was to determine whether thioredoxin 1 (Trx1) mediates the cardioprotective effects of hydrogen sulfide (H2S) in a model of ischemic-induced heart failure (HF). APPROACH AND RESULTS Mice with a cardiac-specific overexpression of a dominant negative mutant of Trx1 and wild-type littermates were subjected to ischemic-induced HF. Treatment with H2S as sodium sulfi...

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