نتایج جستجو برای: جهش NS5B

تعداد نتایج: 3979  

ژورنال: دنیای میکروب ها 2019

سابقه و هدف: ویروس هپاتیت C عامل اصلی هپاتیت مزمن کبدی است که سالیانه باعث مرگ هزاران نفر در دنیا می گردد. پروتئین NS5B، RNA پلی مراز وابسته به RNA است که توسط ژن NS5B کد می شود و در همانندسازی ویروس نقش دارد. از جمله داروهای موثر در درمان این عفونت های ناشی از این ویروس مهارکننده های پروتئین NS5B می باشند. ظهور سویه های مقاوم به این داروها یک مانع بزرگ در موفقیت درمان...

Journal: :Journal of virology 2013
Jing-Tang Huang Ching-Ping Tseng Mei-Huei Liao Shao-Chun Lu Wei-Zhou Yeh Naoya Sakamoto Chuan-Mu Chen Ju-Chien Cheng

Hepatitis C virus (HCV) nonstructural protein 5B (NS5B) is an RNA-dependent RNA polymerase (RdRp) that acts as a key player in the HCV replication complex. Understanding the interplay between the viral and cellular components of the HCV replication complex could provide new insight for prevention of the progression of HCV-associated hepatocellular carcinoma (HCC). In this study, the NS5B protei...

2013
Elizabeth M Quezada Caroline M Kane

The Hepatitis C Virus RNA dependent RNA polymerase, NS5B, is stimulated by the NS5A protein in vitro. To explore this stimulatory mechanism, we compared the activity of a mutant of NS5B containing a deletion of the β-loop region with that of the full length NS5B in response to NS5A. While the NS5A protein does stimulate full length NS5B, NS5A does not stimulate the NS5B deletion mutant during e...

Journal: :The Journal of biological chemistry 2009
Julie Qi Hang Yanli Yang Seth F Harris Vincent Leveque Hannah J Whittington Sonal Rajyaguru Gloria Ao-Ieong Matthew F McCown April Wong Anthony M Giannetti Sophie Le Pogam Francisco Talamás Nick Cammack Isabel Nájera Klaus Klumpp

The binding affinity of four palm and thumb site representative non-nucleoside inhibitors (NNIs) of HCV polymerase NS5B to wild-type and resistant NS5B polymerase proteins was determined, and the influence of RNA binding on NNI binding affinity was investigated. NNIs with high binding affinity potently inhibited HCV RNA polymerase activity and replicon replication. Among the compounds tested, H...

HCV-induced hepatitis is one of the most debilitating diseases. The limited number of anti-HCV drugs and drug-resistance necessitate developing of new scaffolds with different mode of actions. HCV non-structural protein 5B (NS5B) is an attractive target for development of novel inhibitors of HCV replication. In this paper, new N'-arylidene-6-(benzyloxy)-4-oxo-1,4-dihydroquinoline-3-carbohydrazi...

HCV-induced hepatitis is one of the most debilitating diseases. The limited number of anti-HCV drugs and drug-resistance necessitate developing of new scaffolds with different mode of actions. HCV non-structural protein 5B (NS5B) is an attractive target for development of novel inhibitors of HCV replication. In this paper, new N'-arylidene-6-(benzyloxy)-4-oxo-1,4-dihydroquinoline-3-carbohydrazi...

2016
Marleen H M Hessel Adam F Cohen Robert Rissmann

After entering hepatocytes, the viral genome of the hepatitis C virus (HCV) is translated into a single polypeptide. This polypeptide is subsequently cleaved into viral proteins, including non-structural (NS) proteins NS3, NS4A, NS5B and NS5B RNA dependent RNA polymerase (Figure 1) [3, 4]. These viral proteins are essential for viral replication and assembly making them significant targets for ...

2012
Christy M. Hebner Bin Han Katherine M. Brendza Michelle Nash Maisoun Sulfab Yang Tian Magdeleine Hung Wanchi Fung Randall W. Vivian James Trenkle James Taylor Kyla Bjornson Steven Bondy Xiaohong Liu John Link Johan Neyts Roman Sakowicz Weidong Zhong Hengli Tang Uli Schmitz

Tegobuvir (TGV) is a novel non-nucleoside inhibitor (NNI) of HCV RNA replication with demonstrated antiviral activity in patients with genotype 1 chronic HCV infection. The mechanism of action of TGV has not been clearly defined despite the identification of resistance mutations mapping to the NS5B polymerase region. TGV does not inhibit NS5B enzymatic activity in biochemical assays in vitro, s...

2011
Pilar Clemente-Casares Alberto J. López-Jiménez Itxaso Bellón-Echeverría José Antonio Encinar Elisa Martínez-Alfaro Ricardo Pérez-Flores Antonio Mas

Hepatitis C virus (HCV) shows a great geographical diversity reflected in the high number of circulating genotypes and subtypes. The response to HCV treatment is genotype specific, with the predominant genotype 1 showing the lowest rate of sustained virological response. Virally encoded enzymes are candidate targets for intervention. In particular, promising antiviral molecules are being develo...

Journal: :Antiviral chemistry & chemotherapy 2009
Ye Chen Alain Bopda-Waffo Amartya Basu Ramalingam Krishnan Erica Silberstein Deborah R Taylor Tanaji T Talele Payal Arora Neerja Kaushik-Basu

BACKGROUND Hepatitis C virus (HCV) NS5B is an essential component of the viral replication machinery and an important target for antiviral intervention. Aurintricarboxylic acid (ATA), a broad-spectrum antiviral agent, was evaluated and characterized for its anti-NS5B activity in vitro and in HCV replicon cells. METHODS Recombinant NS5B, HCV replicase and Huh-7 cells harbouring the subgenomic ...

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