نتایج جستجو برای: برنامه calm

تعداد نتایج: 65717  

پایان نامه :وزارت علوم، تحقیقات و فناوری - دانشگاه تبریز - دانشکده روانشناسی و علوم تربیتی 1393

هدف پژوهش حاضر بررسی اثربخشی برنامه درمانی مربیگری رفتار و هدایت از طریق مدلسازی(calm program) بر کاهش نشانه های اختلال اضطراب جدایی کودکان 3 تا 7 ساله بود.نمونه پژوهش 5 کودک با اختلال اضطراب جدایی بودند که همراه با مادرشان در12 جلسه درمانی شرکت کردند.طرح پژوهش از نوع تک آزمودنی بود و در آن داده های مربوط به تاثیر مداخله(جلسات درمانی) از طریق مقیاس اضطراب کودکان اسپنس جمع آوری شد.داده های به دس...

Journal: :The Primary Care Companion to The Journal of Clinical Psychiatry 2008

2014
Shinya Rai Hirokazu Tanaka Mai Suzuki Honami Ogoh Yasuhiro Taniguchi Yasuyoshi Morita Takahiro Shimada Akira Tanimura Keiko Matsui Takafumi Yokota Kenji Oritani Kenji Tanabe Toshio Watanabe Yuzuru Kanakura Itaru Matsumura Tsutomu Kume

CALM is implicated in the formation of clathrin-coated vesicles, which mediate endocytosis and intracellular trafficking of growth factor receptors and nutrients. We previously found that CALM-deficient mice suffer from severe anemia due to the impaired clathrin-mediated endocytosis of transferrin receptor in immature erythroblast. However, CALM has been supposed to regulate the growth and surv...

2012
Mai Suzuki Hirokazu Tanaka Akira Tanimura Kenji Tanabe Natsuko Oe Shinya Rai Syunsuke Kon Manabu Fukumoto Kohji Takei Takaya Abe Itaru Matsumura Yuzuru Kanakura Toshio Watanabe

Phosphatidylinositol binding clathrin assembly protein (PICALM), also known as clathrin assembly lymphoid myeloid leukemia protein (CALM), was originally isolated as part of the fusion gene CALM/AF10, which results from the chromosomal translocation t(10;11)(p13;q14). CALM is sufficient to drive clathrin assembly in vitro on lipid monolayers and regulates clathrin-coated budding and the size an...

Journal: :Blood 2013
Amanda E Conway Paula B Scotland Catherine P Lavau Daniel S Wechsler

The t(10;11) chromosomal translocation gives rise to the CALM-AF10 fusion gene and is found in patients with aggressive and difficult-to-treat hematopoietic malignancies. CALM-AF10-driven leukemias are characterized by HOXA gene up-regulation and a global reduction in H3K79 methylation. DOT1L, the H3K79 methyltransferase, interacts with the octapeptide/leucine zipper domain of AF10, and this re...

2013
Daniela A. Sahlender Patrycja Kozik Sharon E. Miller Andrew A. Peden Margaret S. Robinson

CALM (clathrin assembly lymphoid myeloid leukemia protein) is a cargo-selective adaptor for the post-Golgi R-SNAREs VAMPs 2, 3, and 8, and it also regulates the size of clathrin-coated pits and vesicles at the plasma membrane. The present study has two objectives: to determine whether CALM can sort additional VAMPs, and to investigate whether VAMP sorting contributes to CALM-dependent vesicle s...

Journal: :The Iowa Review 1979

Journal: :Haematologica 2006
Roberta La Starza Barbara Crescenzi Alexandre Krause Valentina Pierini Giorgina Specchia Antonella Bardi Rosa Nieddu Cristina Ariola Mauro Nanni Daniela Diverio Anna Aventin Marco Sborgia Massimo F Martelli Stefan K Bohlander Cristina Mecucci

We developed dual-color split fluorescence in situ hybridization (FISH) assays to detect AF10 and/or CALM rearrangements. Among nine cases of acute leukemia with translocation breakpoints at 10p13 and 11q14-21, a CALM/AF10 rearrangement was found in seven and was confirmed by reverse transcriptase polymerase chain reaction (RT-PCR) in all. In 2/7 cases, FISH detected CALM/AF10 in extramedullary...

Journal: :Molecular biology of the cell 1999
F Tebar S K Bohlander A Sorkin

The clathrin assembly lymphoid myeloid leukemia (CALM) gene encodes a putative homologue of the clathrin assembly synaptic protein AP180. Hence the biochemical properties, the subcellular localization, and the role in endocytosis of a CALM protein were studied. In vitro binding and coimmunoprecipitation demonstrated that the clathrin heavy chain is the major binding partner of CALM. The bulk of...

Journal: :Genes, chromosomes & cancer 2003
Daniel S Wechsler Lars D Engstrom Brian M Alexander David G Motto Diane Roulston

Rearrangements involving the MLL gene at chromosome band 11q23 are common in infant acute myeloid leukemias (AMLs). We recently encountered an infant patient with rapidly progressive AML whose leukemic cells harbored a previously undescribed MLL rearrangement involving an inversion of 11q [inv(11)(q14q23)]. We used panhandle PCR to determine that this rearrangement juxtaposed the MLL (Mixed-Lin...

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