نتایج جستجو برای: vascular smooth muscle cell

تعداد نتایج: 2156167  

Journal: :Atherosclerosis 2021

Background and Aims: Aim: Pathological vascular remodeling is the underlying cause of main cardiovascular diseases (CVD) including atherosclerosis abdominal aortic aneurysm (AAA). Diminished smooth muscle cell (VSMC) content a key mechanism atherosclerotic plaques AAA instability. We hypothesized that Galectin-1 (Gal-1) could exert protective effects in as previously demonstrated different chro...

Objective(s): To investigate the role of autophagy in advanced glycation end products (AGEs)-induced proliferation and migration in rat vascular smooth muscle cells (VSMCs).Materials and Methods: After culture, VSMCs were treated with 0, 1, 10, and 100 μg/ml concentrations of AGEs. Autophagy specific protein light chain 3 (LC3)-I/II was determined by western blotting, autophagosomes were observ...

Journal: :Hypertension 2017
Kazuhiro Nakao Koichiro Kuwahara Toshio Nishikimi Yasuaki Nakagawa Hideyuki Kinoshita Takeya Minami Yoshihiro Kuwabara Chinatsu Yamada Yuko Yamada Takeshi Tokudome Chiaki Nagai-Okatani Naoto Minamino Yoko M Nakao Shinji Yasuno Kenji Ueshima Masakatsu Sone Takeshi Kimura Kenji Kangawa Kazuwa Nakao

We previously reported the secretion of C-type natriuretic peptide (CNP) from vascular endothelial cells and proposed the existence of a vascular natriuretic peptide system composed of endothelial CNP and smooth muscle guanylyl cyclase-B (GC-B), the CNP receptor, and involved in the regulation of vascular tone, remodeling, and regeneration. In this study, we assessed the functional significance...

2017
Wei-Wei Li Hai-Yue Wang Xi Nie Ya-Bin Liu Mei Han Bing-Hui Li

Tumor vessels often lack the smooth muscle layer, and the instability is conducive to tumor invasion and metastasis. The effect of tumor microenvironment on vascular smooth muscle cells needs to be explored. In the present study, we examined the density of the tumor vessels in human colorectal cancer tissues, and used the tumor conditioned medium of human colorectal cancer HT29 cells to mimic t...

Journal: :Trends in cardiovascular medicine 2010
Lihong Chen Guangrui Yang Xiufeng Xu Gregory Grant John A Lawson Mohammad Bohlooly-Y Garret A FitzGerald

BACKGROUND Global deletion of microsomal prostaglandin E synthase 1 (mPGES-1) in mice attenuates the response to vascular injury without a predisposition to thrombogenesis or hypertension. However, enzyme deletion results in cell-specific differential use by prostaglandin synthases of the accumulated prostaglandin H(2) substrate. Here, we generated mice deficient in mPGES-1 in vascular smooth m...

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