نتایج جستجو برای: vaccine escape mutations

تعداد نتایج: 308143  

Journal: :The Journal of clinical investigation 2009
Eva Dazert Christoph Neumann-Haefelin Stéphane Bressanelli Karen Fitzmaurice Julia Kort Jörg Timm Susan McKiernan Dermot Kelleher Norbert Gruener John E Tavis Hugo R Rosen Jaqueline Shaw Paul Bowness Hubert E Blum Paul Klenerman Ralf Bartenschlager Robert Thimme

There is an association between expression of the MHC class I molecule HLA-B27 and protection following human infection with either HIV or HCV. In both cases, protection has been linked to HLA-B27 presentation of a single immunodominant viral peptide epitope to CD8+ T cells. If HIV mutates the HLA-B27-binding anchor of this epitope to escape the protective immune response, the result is a less-...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2013
Sophie A Valkenburg Stephanie Gras Carole Guillonneau Lauren A Hatton Nicola A Bird Kelly-Anne Twist Hanim Halim David C Jackson Anthony W Purcell Stephen J Turner Peter C Doherty Jamie Rossjohn Katherine Kedzierska

A reverse-genetics approach has been used to probe the mechanism underlying immune escape for influenza A virus-specific CD8(+) T cells responding to the immunodominant D(b)NP366 epitope. Engineered viruses with a substitution at a critical residue (position 6, P6M) all evaded recognition by WT D(b)NP366-specific CD8(+) T cells, but only the NPM6I and NPM6T mutants altered the topography of a k...

Journal: :Journal of virology 2012
Cindy Luongo Christine C Winter Peter L Collins Ursula J Buchholz

Human respiratory syncytial virus (RSV) is the most important viral cause of serious pediatric respiratory illness worldwide. Currently, the most promising live-attenuated vaccine candidate is a temperature-sensitive (ts) cDNA-derived virus named rA2cp248/404/1030ΔSH, in reference to its set of attenuating mutations. In a previous clinical study, more than one-third of postvaccination nasal was...

2018
Xingui Tian Hongling Qiu Zhichao Zhou Shouli Wang Ye Fan Xiao Li Ruiai Chu Haitao Li Rong Zhou Hui Wang

Human adenovirus type 4 (HAdV-4) is an epidemic virus that contributes to serious acute respiratory disease (ARD) in both pediatric and adult patients. However, no licensed drug or vaccine is currently available to the civilian population. The identification of neutralizing epitopes of HAdV-4 should allow the development of a novel antiviral vaccine and a novel gene transfer vector, and an effe...

2011
Sayuri Seki Tetsuro Matano

Cytotoxic T lymphocyte (CTL) responses exert a suppressive effect on HIV and simian immunodeficiency virus (SIV) replication. Under the CTL pressure, viral CTL escape mutations are frequently selected with viral fitness costs. Viruses with such CTL escape mutations often need additional viral genome mutations for recovery of viral fitness. Persistent HIV/SIV infection sometimes shows replacemen...

Journal: :Intervirology 2006
Syed Naqui Kazim Shiv Kumar Sarin Barjesh Chander Sharma Luqman Ahmad Khan Seyed Ehtesham Hasnain

BACKGROUND Besides vaccine escape or immune escape hepatitis B virus (HBV) mutants, naturally occurring and drug-induced mutations have been reported in the surface gene (S-gene) of HBV. AIM To investigate the frequency and profile of naturally occurring S-gene mutants and the influence of long-term lamivudine therapy in patients with chronic hepatitis B (CHB). MATERIALS AND METHODS 57 pati...

Journal: :PLoS Pathogens 2007
Zabrina L Brumme Chanson J Brumme David Heckerman Bette T Korber Marcus Daniels Jonathan Carlson Carl Kadie Tanmoy Bhattacharya Celia Chui James Szinger Theresa Mo Robert S Hogg Julio S. G Montaner Nicole Frahm Christian Brander Bruce D Walker P. Richard Harrigan

Despite the formidable mutational capacity and sequence diversity of HIV-1, evidence suggests that viral evolution in response to specific selective pressures follows generally predictable mutational pathways. Population-based analyses of clinically derived HIV sequences may be used to identify immune escape mutations in viral genes; however, prior attempts to identify such mutations have been ...

Journal: :PLoS Computational Biology 2007
David C. Nickle Morgane Rolland Mark A. Jensen Sergei L. Kosakovsky Pond Wenjie Deng Mark Seligman David Heckerman James I. Mullins Nebojsa Jojic

The ability of human immunodeficiency virus type 1 (HIV-1) to develop high levels of genetic diversity, and thereby acquire mutations to escape immune pressures, contributes to the difficulties in producing a vaccine. Possibly no single HIV-1 sequence can induce sufficiently broad immunity to protect against a wide variety of infectious strains, or block mutational escape pathways available to ...

Journal: :Journal of virology 2012
Jonathan M Carlson Chanson J Brumme Eric Martin Jennifer Listgarten Mark A Brockman Anh Q Le Celia K S Chui Laura A Cotton David J H F Knapp Sharon A Riddler Richard Haubrich George Nelson Nico Pfeifer Charles E Deziel David Heckerman Richard Apps Mary Carrington Simon Mallal P Richard Harrigan Mina John Zabrina L Brumme

HLA class I-associated polymorphisms identified at the population level mark viral sites under immune pressure by individual HLA alleles. As such, analysis of their distribution, frequency, location, statistical strength, sequence conservation, and other properties offers a unique perspective from which to identify correlates of protective cellular immunity. We analyzed HLA-associated HIV-1 sub...

Journal: :Environmental Health Perspectives 2001
BM Weinhold

The ability of human immunodeficiency virus type 1 (HIV-1) to develop high levels of genetic diversity, and thereby acquire mutations to escape immune pressures, contributes to the difficulties in producing a vaccine. Possibly no single HIV-1 sequence can induce sufficiently broad immunity to protect against a wide variety of infectious strains, or block mutational escape pathways available to ...

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