نتایج جستجو برای: syn dienvelope

تعداد نتایج: 6083  

1999
Livio Ricciulli Patrick Lincoln Pankaj Kakkar

The TCP SYN ooding denial-of-service attack pointed out a weakness of then-current Internet protocols. There have since been many proposals to defend against SYN ooding, some requiring signiicant changes to TCP. Several solutions attempting to resolve the TCP weakness are now generally available. We document these existing solutions and qualitatively compare them. We reene the analysis of the r...

2012
Anna E. Cartier Kiren Ubhi Brian Spencer Ruben A. Vazquez-Roque Kori Ann Kosberg Lawrence Fourgeaud Priya Kanayson Christina Patrick Edward Rockenstein Gentry N. Patrick Eliezer Masliah

Parkinson's disease (PD) is a progressive neurodegenerative disorder caused by genetic and environmental factors. Abnormal accumulation and aggregation of alpha-synuclein (a-syn) within neurons, and mutations in the a-syn and UCH-L1 genes have been shown to play a role in the pathogenesis of PD. In light of recent reports suggesting an interaction between a-synuclein and UCH-L1, we investigated...

2011
Xiaohui Cang Jiří Šponer Thomas E. Cheatham

Guanine-rich DNA sequences tend to form four-stranded G-quadruplex structures. Characteristic glycosidic conformational patterns along the G-strands, such as the 5'-syn-anti-syn-anti pattern observed with the Oxytricha nova telomeric G-quadruplexes, have been well documented. However, an explanation for these featured glycosidic patterns has not emerged. This work presents MD simulation and fre...

2011
Eitan Israeli Eugenia Yakunin Yonaton Zarbiv Amir Hacohen-Solovich Haya Kisos Virginie Loeb Michal Lichtenstein Tziona Ben-Gedalya Ofra Sabag Eli Pikarsky Haya Lorberboum-Galski Ronit Sharon

Alpha Synuclein (α-Syn) is a protein implicated in mechanisms of neuronal degeneration in Parkinson's disease (PD). α-Syn is primarily a neuronal protein, however, its expression is found in various tumors including ovarian, colorectal and melanoma tumors. It has been hypothesized that neurodegeneration may share common mechanisms with oncogenesis. We tested whether α-Syn expression affects tum...

2011
Nan Jiang Dayong Xue Hongxiang Han

The genus Biston Leach, 1815 is reviewed for China. Seventeen species are recognized, of which Biston mediolatasp. n. is described. Biston pustulata (Warren, 1896) and Biston panterinariaexanthemata (Moore, 1888) are newly recorded for China. The following new synonyms are established: Biston suppressaria suppressaria (Guenée, 1858) (= Biston suppressaria benescripta (Prout, 1915), syn. n. = Bi...

2018
Seiji Kaji Takakuni Maki Hisanori Kinoshita Norihito Uemura Takashi Ayaki Yasuhiro Kawamoto Takahiro Furuta Makoto Urushitani Masato Hasegawa Yusuke Kinoshita Yuichi Ono Xiaobo Mao Tran H. Quach Kazuhiro Iwai Valina L. Dawson Ted M. Dawson Ryosuke Takahashi

Glial cytoplasmic inclusions (GCIs), commonly observed as α-synuclein (α-syn)-positive aggregates within oligodendrocytes, are the pathological hallmark of multiple system atrophy. The origin of α-syn in GCIs is uncertain; there is little evidence of endogenous α-syn expression in oligodendrocyte lineage cells, oligodendrocyte precursor cells (OPCs), and mature oligodendrocytes (OLGs). Here, ba...

2011
Eliezer Masliah Edward Rockenstein Michael Mante Leslie Crews Brian Spencer Anthony Adame Christina Patrick Margarita Trejo Kiren Ubhi Troy T. Rohn Sarah Mueller-Steiner Peter Seubert Robin Barbour Lisa McConlogue Manuel Buttini Dora Games Dale Schenk

Dementia with Lewy bodies (DLB) and Parkinson's Disease (PD) are common causes of motor and cognitive deficits and are associated with the abnormal accumulation of alpha-synuclein (α-syn). This study investigated whether passive immunization with a novel monoclonal α-syn antibody (9E4) against the C-terminus (CT) of α-syn was able to cross into the CNS and ameliorate the deficits associated wit...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
Youngshin Lim Victoria M Kehm Edward B Lee James H Soper Chi Li John Q Trojanowski Virginia M-Y Lee

Abnormally accumulated α-synuclein (α-syn) is a pathological hallmark of Lewy body-related disorders such as Parkinson's disease (PD) and dementia with Lewy body disease (DLB). However, it is not well understood whether and how abnormal accumulation of α-syn leads to cognitive impairment or dementia in PD and DLB. Furthermore, it is not known whether targeted removal of α-syn pathology can reve...

2015
Mathieu Bourdenx Sandra Dovero Michel Engeln Simone Bido Matthieu F. Bastide Nathalie Dutheil Isabel Vollenweider Laetitia Baud Camille Piron Virginie Grouthier Thomas Boraud Grégory Porras Qin Li Veerle Baekelandt Dieter Scheller Anne Michel Pierre-Olivier Fernagut François Georges Grégoire Courtine Erwan Bezard Benjamin Dehay

INTRODUCTION Parkinson's disease (PD) is a progressive neurodegenerative disorder characterized by the loss of dopaminergic neurons as well as the presence of proteinaceous inclusions named Lewy bodies. α-synuclein (α-syn) is a major constituent of Lewy bodies, and the first disease-causing protein characterized in PD. Several α-syn-based animal models of PD have been developed to investigate t...

Journal: :PLoS ONE 2008
Sara Batelli Diego Albani Raffaela Rametta Letizia Polito Francesca Prato Marzia Pesaresi Alessandro Negro Gianluigi Forloni

BACKGROUND Parkinson's disease (PD) is a neurodegenerative pathology whose molecular etiopathogenesis is not known. Novel contributions have come from familial forms of PD caused by alterations in genes with apparently unrelated physiological functions. The gene coding for alpha-synuclein (alpha-syn) (PARK1) has been investigated as alpha-syn is located in Lewy bodies (LB), intraneuronal inclus...

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