نتایج جستجو برای: myc

تعداد نتایج: 16295  

Journal: :Genes & development 2000
B A Malynn I M de Alboran R C O'Hagan R Bronson L Davidson R A DePinho F W Alt

Members of the myc family of cellular oncogenes have been implicated as transcriptional regulators in pathways that govern cellular proliferation and death. In addition, N-myc and c-myc are essential for completion of murine embryonic development. However, the basis for the evolutionary conservation of myc gene family has remained unclear. To elucidate this issue, we have generated mice in whic...

Journal: :Biochemistry 2006
Robert A Burton Sampo Mattila Elizabeth J Taparowsky Carol Beth Post

B-Myc is an endogenous, N-terminal homologue of transcription factor c-Myc that lacks the C-terminal DNA binding and protein dimerization domain of c-Myc. Clinical mutations in the c-Myc N-terminal region, and the subsequent misregulation of Myc, are implicated in the development of numerous human cancers. Myc functions to both activate and repress transcription by associating with multiple bin...

Journal: :The Journal of biological chemistry 2008
Maryam Niapour Yongmao Yu Stuart A Berger

The c-Myc transcription factor is commonly dysregulated in cancer. c-Myc also sensitizes cells to apoptosis induced by a variety of toxic events. c-Myc turnover is rapid and mediated by the proteasome and intracellular calpains. Therefore, c-Myc accumulation could contribute to cell death associated with protease inhibitors. We investigated the response of c-Myc-positive and c-Myc-negative rat ...

2014
Philipp Raffeiner Ruth Röck Andrea Schraffl Markus Hartl Jonathan R. Hart Kim D. Janda Peter K. Vogt Eduard Stefan Klaus Bister

The oncogenic bHLH-LZ transcription factor Myc forms binary complexes with its binding partner Max. These and other bHLH-LZ-based protein-protein interactions (PPI) in the Myc-Max network are essential for the physiological and oncogenic activities of Myc. We have generated a genetically determined and highly specific protein-fragment complementation assay based on Renilla luciferase to analyze...

2017
Julum Nwanze Momin T. Siddiqui Keith A. Stevens Debra Saxe Cynthia Cohen

MYC is the proto-oncogene classically associated with Burkitt lymphoma (BL) located at chromosomal locus 8q24. Rearrangements of MYC are seen in nearly 100% of BL but have been reported in 3-16% of diffuse large B-cell lymphomas (DLBCLs). Rearrangements of MYC are tested for by flourescence in situ hybridization (FISH). In this study, we compared immunohistochemistry (IHC) using a monoclonal an...

2018
Gilbert Bigras Wei-Feng Dong Sarah Canil Raymond Lai Didier Morel Paul E. Swanson Iyare Izevbaye

A new automated MYC IHC classifier based on bivariate logistic regression is presented. The predictor relies on image analysis developed with the open-source ImageJ platform. From a histologic section immunostained for MYC protein, 2 dimensionless quantitative variables are extracted: (a) relative distance between nuclei positive for MYC IHC based on euclidean minimum spanning tree graph and (b...

Journal: :Molecular cell 2015
Lance R Thomas Qingguo Wang Brian C Grieb Jason Phan Audra M Foshage Qi Sun Edward T Olejniczak Travis Clark Soumyadeep Dey Shelly Lorey Bethany Alicie Gregory C Howard Bryan Cawthon Kevin C Ess Christine M Eischen Zhongming Zhao Stephen W Fesik William P Tansey

MYC is an oncoprotein transcription factor that is overexpressed in the majority of malignancies. The oncogenic potential of MYC stems from its ability to bind regulatory sequences in thousands of target genes, which depends on interaction of MYC with its obligate partner, MAX. Here, we show that broad association of MYC with chromatin also depends on interaction with the WD40-repeat protein WD...

2016
Luana D'Artista Andrea Bisso Andrea Piontini Mirko Doni Alessandro Verrecchia Theresia R. Kress Marco J. Morelli Giannino Del Sal Bruno Amati Stefano Campaner

The c-myc proto-oncogene is activated by translocation in Burkitt's lymphoma and substitutions in codon 58 stabilize the Myc protein or augment its oncogenic potential. In wild-type Myc, phosphorylation of Ser 62 and Thr 58 provides a landing pad for the peptidyl prolyl-isomerase Pin1, which in turn promotes Ser 62 dephosphorylation and Myc degradation. However, the role of Pin1 in Myc-induced ...

Journal: :Cancer research 1988
B E Johnson R W Makuch A D Simmons A F Gazdar D Burch A W Cashell

Tumor specimens procured from 38 different small cell lung cancer patients were studied for DNA amplification of the myc family of protooncogenes (c-myc, N-myc, and L-myc). Six of the 38 specimens (16%) had 4-fold or greater myc family DNA amplification (N-myc in 4 and L-myc in 2). All 6 tumors with amplification came from patients who had received combination chemotherapy. The myc family gene ...

Journal: :Haematologica 2014
Sietse M Aukema Markus Kreuz Christian W Kohler Maciej Rosolowski Dirk Hasenclever Michael Hummel Ralf Küppers Dido Lenze German Ott Christiane Pott Julia Richter Andreas Rosenwald Monika Szczepanowski Carsten Schwaenen Harald Stein Heiko Trautmann Swen Wessendorf Lorenz Trümper Markus Loeffler Rainer Spang Philip M Kluin Wolfram Klapper Reiner Siebert

Chromosomal translocations affecting the MYC oncogene are the biological hallmark of Burkitt lymphomas but also occur in a subset of other mature B-cell lymphomas. If accompanied by a chromosomal break targeting the BCL2 and/or BCL6 oncogene these MYC translocation-positive (MYC(+)) lymphomas are called double-hit lymphomas, otherwise the term single-hit lymphomas is applied. In order to charac...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید