نتایج جستجو برای: mitokatp

تعداد نتایج: 121  

2012
D. Brian Foster Alice S. Ho Jasma Rucker Anders O. Garlid Ling Chen Agnieszka Sidor Keith D. Garlid Brian O’Rourke

Journal: :Molecular medicine reports 2014
Yanyan Hu Li Li Wenbin Yin Lin Shen Beian You Haiqing Gao

The aim of the present study was to examine the protective effect of proanthocyanidins anoxia-reoxygenation injury of myocardial cells and its association with phosphatidylinositol-3-kinase/Akt and glycogen synthase kinase (PI3K/Akt/GSK)-3β and ATP-sensitive potassium channels. Neonatal rat myocardial cells were cultured and an anoxia-reoxygenation model was established following pretreatment w...

Journal: :American journal of physiology. Heart and circulatory physiology 2006
Anastasia Andrukhiv Alexandre D Costa Ian C West Keith D Garlid

Opening the mitochondrial ATP-sensitive K(+) channel (mitoK(ATP)) increases levels of reactive oxygen species (ROS) in cardiomyocytes. This increase in ROS is necessary for cardioprotection against ischemia-reperfusion injury; however, the mechanism of mitoK(ATP)-dependent stimulation of ROS production is unknown. We examined ROS production in suspensions of isolated rat heart and liver mitocho...

Journal: :American journal of physiology. Heart and circulatory physiology 2004
Qining Qin Xi-Ming Yang Lin Cui Stuart D Critz Michael V Cohen Natasha C Browner Thomas M Lincoln James M Downey

Exogenous nitric oxide (NO) triggers a preconditioning-like effect in heart via a pathway that is dependent on reactive oxygen species. This study examined the signaling pathway by which the NO donor S-nitroso-N-acetylpenicillamine (SNAP, 2 microM) triggers its anti-infarct effect. Isolated rabbit hearts experienced 30 min of regional ischemia and 120 min of subsequent reperfusion. Infarct size...

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