نتایج جستجو برای: glycine active site

تعداد نتایج: 780009  

Journal: :The Journal of pharmacology and experimental therapeutics 1998
E Viu A Zapata J L Capdevila L H Fossom P Skolnick R Trullas

Activation of N-methyl-D-aspartate (NMDA) receptors is known to produce arachidonic acid release, which has been implicated in excitotoxicity. Antagonists and partial agonists at the glycine site of the NMDA receptor, despite exhibiting functional differences in electrophysiological studies, inhibit glutamate-induced neurotoxicity and ischemia-induced neurodegeneration. The objective of this st...

Journal: :Plant physiology 1979
D J Oliver

Isolated soybean leaf mesophyll cells decarboxylated exogenously added [1-(14)C]glycolate and [1-(14)C]glycine in the dark. The rate of CO(2) release from glycine was inhibited over 90% by isonicotinic acid hydrazide and about 80% by KCN, two inhibitors of the glycine to serine plus CO(2) reaction. The release of CO(2) from glycolate was inhibited by less than 50% under the same conditions. Thi...

Journal: :The Journal of biological chemistry 2012
Eloisa Carta Seo-Kyung Chung Victoria M James Angela Robinson Jennifer L Gill Nathalie Remy Jean-François Vanbellinghen Cheney J G Drew Sophie Cagdas Duncan Cameron Frances M Cowan Mireria Del Toro Gail E Graham Adnan Y Manzur Amira Masri Serge Rivera Emmanuel Scalais Rita Shiang Kate Sinclair Catriona A Stuart Marina A J Tijssen Grahame Wise Sameer M Zuberi Kirsten Harvey Brian R Pearce Maya Topf Rhys H Thomas Stéphane Supplisson Mark I Rees Robert J Harvey

Hereditary hyperekplexia or startle disease is characterized by an exaggerated startle response, evoked by tactile or auditory stimuli, leading to hypertonia and apnea episodes. Missense, nonsense, frameshift, splice site mutations, and large deletions in the human glycine receptor α1 subunit gene (GLRA1) are the major known cause of this disorder. However, mutations are also found in the genes...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1998
L C Anson P E Chen D J Wyllie D Colquhoun R Schoepfer

The NMDA type of ligand-gated glutamate receptor requires the presence of both glutamate and glycine for gating. These receptors are hetero-oligomers of NR1 and NR2 subunits. Previously it was thought that the binding sites for glycine and glutamate were formed by residues on the NR1 subunit. Indeed, it has been shown that the effects of glycine are controlled by residues on the NR1 subunit, an...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1981
M A Zarbin J K Wamsley M J Kuhar

Glycine receptors have been localized by autoradiography in the rat central nervous system (CNS) using [3H]strychnine. The gross distribution of receptors is in excellent accord with the distribution determined by filtration binding assays. Specifically, the density of glycine receptors is greatest in the gray matter of the spinal cord and decreases progressively in regions more rostral in the ...

2016
Rong Hu Juan Chen Brendan Lujan Ruixue Lei Mi Zhang Zefen Wang Mingxia Liao Zhiqiang Li Yu Wan Fang Liu Hua Feng Qi Wan

Ionotropic activation of NMDA receptors (NMDARs) requires agonist glutamate and co-agonist glycine. Here we show that glycine enhances the activation of cell survival-promoting kinase Akt in cultured cortical neurons in which both the channel activity of NMDARs and the glycine receptors are pre-inhibited. The effect of glycine is reduced by shRNA-mediated knockdown of GluN2A subunit-containing ...

Journal: :The American journal of psychiatry 2007
Robert W Buchanan Daniel C Javitt Stephen R Marder Nina R Schooler James M Gold Robert P McMahon Uriel Heresco-Levy William T Carpenter

OBJECTIVE Patients with schizophrenia frequently present with negative symptoms and cognitive impairments for which no effective treatments are known. Agents that act at the glycine site of the N-methyl-D-aspartic acid (NMDA) glutamatergic receptor have been suggested as promising treatments for moderate to severe negative symptoms and cognitive impairments. METHOD The Cognitive and Negative ...

Journal: :The Journal of pharmacology and experimental therapeutics 1997
A Pittaluga D Vaccari M Raiteri

Some putative cognition enhancers (oxiracetam, aniracetam and D-cycloserine) were previously shown to prevent the kynurenic acid antagonism of the N-methyl-D-aspartate (NMDA)-evoked norepinephrine (NE) release in rat hippocampal slices. This functional in vitro assay was further characterized in the present work. D-Serine, a glutamate coagonist at the NMDA receptor glycine site, concentration-d...

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