نتایج جستجو برای: fti

تعداد نتایج: 436  

Journal: :The Journal of clinical endocrinology and metabolism 2004
Yair Liel

Thyroidectomy (TX) is no longer the preferred choice for the therapy of hyperthyroid Graves' disease but is an alternative in patients who are noncompliant with or have reactions to antithyroid drugs, have moderate to severe ophthalmopathy, have large goiters, or who refuse (131)I therapy and/or long-term antithyroid drug therapy. Seventeen clinically and biochemically severely thyrotoxic patie...

Journal: :Blood 2005
Bin Zhang John Groffen Nora Heisterkamp

Resistance to cytotoxic drugs frequently emerges during treatment of leukemia with conventional chemotherapy. New classes of anticancer drugs, such as the farnesyltransferase inhibitors (FTIs), show therapeutic promise, but whether cells will easily develop resistance against them is not known. Here, we grew breakpoint cluster region/Abelson murine leukemia (Bcr/Abl) P190 lymphoblasts on stroma...

Journal: :Blood 2001
M A Morgan O Dolp C W Reuter

Disruption of the RAS-to-mitogen-activated protein kinase (MAPK/ERK) signaling pathway, either directly through activating RAS gene mutations or indirectly through other genetic aberrations, plays an important role in the molecular pathogenesis of myeloid leukemias. Constitutive activation of ERK-1/2 and MEK-1/2, which elicit oncogenic transformation in fibroblasts, has recently been observed i...

Journal: :Molecular pharmacology 2005
Laura M Bruzek Jenny N Poynter Scott H Kaufmann Alex A Adjei

Farnesyl protein transferase inhibitors (FTIs) have demonstrated clinical activity in certain solid tumors and hematological malignancies. Little is known about mechanisms of resistance to these agents. To provide a basis for better understanding FTI resistance, the colorectal carcinoma cell line HCT 116 was selected by stepwise exposure to increasing 4-(2-(4-(8-chloro-3,10-dibromo-6,11-dihydro...

Journal: :Cancer research 2001
W C Rose F Y Lee C R Fairchild M Lynch T Monticello R A Kramer V Manne

BMS-214662 is a potent and selective inhibitor of farnesyltransferase (FTI). In rodent fibroblasts transformed by oncogenes, BMS-214662 reversed the H-Ras-transformed phenotype but not that of K-Ras or other oncogenes. In soft agar growth assays, BMS-214662 showed good potency in inhibiting H-ras-transformed rodent cells, A2780 human ovarian carcinoma tumor cells, and HCT-116 human colon carcin...

Journal: :The Journal of pharmacology and experimental therapeutics 2011
Wen Yang Marina Yamada Yoshiaki Tamura Kyungho Chang Ji Mao Lin Zou Yan Feng Kotaro Kida Marielle Scherrer-Crosbie Wei Chao Fumito Ichinose Yong-Ming Yu Alan J Fischman Ronald G Tompkins Shanglong Yao Masao Kaneki

Treatment with statins, inhibitors of HMG-CoA reductase, extends the survival of septic mice. However, the molecular mechanisms underlying the cholesterol-lowering, independent beneficial effects of statins in sepsis are poorly understood. The inhibition of protein isoprenylation, namely farnesylation and geranylgeranylation, has been proposed as a mediator of the pleiotropic protective effects...

Journal: :Brazilian Archives of Biology and Technology 1998

Journal: :Jurnal SITECH : Sistem Informasi dan Teknologi 2019

Journal: :PloS one 2015
Jennifer Wegrzyk Alexandre Fouré Yann Le Fur Nicola A Maffiuletti Christophe Vilmen Maxime Guye Jean-Pierre Mattei Nicolas Place David Bendahan Julien Gondin

Conventional (CONV) neuromuscular electrical stimulation (NMES) (i.e., short pulse duration, low frequencies) induces a higher energetic response as compared to voluntary contractions (VOL). In contrast, wide-pulse, high-frequency (WPHF) NMES might elicit--at least in some subjects (i.e., responders)--a different motor unit recruitment compared to CONV that resembles the physiological muscle ac...

2001
William C. Rose Francis Y. F. Lee Craig R. Fairchild Mark Lynch Thomas Monticello Robert A. Kramer Veeraswamy Manne

BMS-214662 is a potent and selective inhibitor of farnesyltransferase (FTI). In rodent fibroblasts transformed by oncogenes, BMS-214662 reversed the H-Ras-transformed phenotype but not that of K-Ras or other oncogenes. In soft agar growth assays, BMS-214662 showed good potency in inhibiting H-ras-transformed rodent cells, A2780 human ovarian carcinoma tumor cells, and HCT-116 human colon carcin...

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