نتایج جستجو برای: cerebellar ataxias

تعداد نتایج: 26906  

Journal: :Neuroscience letters 2012
Zhao Chen Xiaohui Li Beisha Tang Junling Wang Yuting Shi Zhanfang Sun Li Zhang Qian Pan Kun Xia Hong Jiang

Autosomal dominant cerebellar ataxias (ADCAs), genetically classified into spinocerebellar ataxias (SCAs), are a highly heterogeneous group of neurodegenerative disorders. Recently, mutations in the fibroblast growth factor 14 gene (FGF14) have been reported to cause SCA27 subtype. To evaluate the frequency of FGF14 mutations in mainland of China, we performed molecular genetic analysis in 67 u...

Journal: :Cell 2006
Janghoo Lim Tong Hao Chad Shaw Akash J. Patel Gábor Szabó Jean-François Rual C. Joseph Fisk Ning Li Alex Smolyar David E. Hill Albert-László Barabási Marc Vidal Huda Y. Zoghbi

Many human inherited neurodegenerative disorders are characterized by loss of balance due to cerebellar Purkinje cell (PC) degeneration. Although the disease-causing mutations have been identified for a number of these disorders, the normal functions of the proteins involved remain, in many cases, unknown. To gain insight into the function of proteins involved in PC degeneration, we developed a...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2009
Esther B E Becker Peter L Oliver Maike D Glitsch Gareth T Banks Francesca Achilli Andrea Hardy Patrick M Nolan Elizabeth M C Fisher Kay E Davies

The hereditary ataxias are a complex group of neurological disorders characterized by the degeneration of the cerebellum and its associated connections. The molecular mechanisms that trigger the loss of Purkinje cells in this group of diseases remain incompletely understood. Here, we report a previously undescribed dominant mouse model of cerebellar ataxia, moonwalker (Mwk), that displays motor...

Journal: :Neuron 2002
Sandrine Humbert Frédéric Saudou

Transcriptional dysregulation appears as an emerging and unifying pathogenic mechanism in polyQ neurodegenerative disorders such as Spinocerebellar ataxias and Huntington's disease. It is unclear how cell death specificity occurs in these diseases. In this issue of Neuron, link polymerase II, a general component of the transcriptional machinery, to PQBP-1, a cerebellar enriched protein, thus pr...

Journal: :The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques 2003
E K Tan

Autosomal dominant cerebellar ataxias, frequently referred to as spinocerebellar ataxias (SCAs) have been under intense scientific research limelight since expansions of coded CAG trinucleotide repeats were demonstrated to cause several dominantly inherited SCAs. The number of new SCA loci has expanded dramatically in recent years. At least ten genes have been identified for SCAs 1, 2, 3, 6, 7,...

Journal: :American journal of human genetics 2015
Wolfgang M Schmidt S Lane Rutledge Rebecca Schüle Benjamin Mayerhofer Stephan Züchner Eugen Boltshauser Reginald E Bittner

Hereditary ataxias comprise a group of genetically heterogeneous disorders characterized by clinically variable cerebellar dysfunction and accompanied by involvement of other organ systems. The molecular underpinnings for many of these diseases are widely unknown. Previously, we discovered the disruption of Scyl1 as the molecular basis of the mouse mutant mdf, which is affected by neurogenic mu...

Journal: :Journal of medical genetics 2004
P N Valdmanis A A Simões Lopes F Gros-Louis J D Stewart G A Rouleau N Dupré

T he recent barrage of linkage assignments and gene discoveries has confirmed the clinical and genetic heterogeneity of ataxic diseases. They all share the prototypic feature of difficulty in walking though many additionally present dysarthria, spasticity, retinopathy, and other neurological symptoms. Broad subgroups of ataxias and related diseases exist including spinocerebellar and spastic at...

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