نتایج جستجو برای: cardiac fibrosis

تعداد نتایج: 376764  

2014
Kongkiat Chaikriangkrai Juan C Lopez-Mattei Homam Ibrahim Stephen H Little William Zoghbi Miguel A Quinones Gerald Lawrie Dipan J Shah

Background Cardiac MRI has been shown to be the gold standard for noninvasive assessment of myocardial fibrosis. The extent of fibrosis has been associated with worse outcomes in patients undergoing CABG surgery. The presence of fibrosis and associated clinical features of patients with chronic mitral regurgitation undergoing mitral valve repair have not been described. The objective of this st...

2012
Melissa Touvron Brigitte Escoubet Mathias Mericskay Aude Angelini Luciane Lamotte Maria Paola Santini Nadia Rosenthal Dominique Daegelen David Tuil Jean-François Decaux

Cardiac fibrosis is critically involved in the adverse remodeling accompanying dilated cardiomyopathies (DCMs), which leads to cardiac dysfunction and heart failure (HF). Connective tissue growth factor (CTGF), a profibrotic cytokine, plays a key role in this deleterious process. Some beneficial effects of IGF1 on cardiomyopathy have been described, but its potential role in improving DCM is le...

Journal: :Circulation 2012
Navin K Kapur Szuhuei Wilson Adil A Yunis Xiaoying Qiao Emily Mackey Vikram Paruchuri Corey Baker Mark J Aronovitz S Ananth Karumanchi Michelle Letarte David A Kass Michael E Mendelsohn Richard H Karas

BACKGROUND Heart failure is a major cause of morbidity and mortality worldwide. The ubiquitously expressed cytokine transforming growth factor-β1 (TGFβ1) promotes cardiac fibrosis, an important component of progressive heart failure. Membrane-associated endoglin is a coreceptor for TGFβ1 signaling and has been studied in vascular remodeling and preeclampsia. We hypothesized that reduced endogli...

2018
Yue Ji Ming Qiu Yejiao Shen Li Gao Yaqing Wang Wei Sun Xinli Li Yan Lu Xiangqing Kong

MicroRNA (miRNA/miR) dysregulation has been reported to be fundamental in the development and progression of cardiac hypertrophy and fibrosis. In the present study, miR-327 levels in fibroblasts were increased in response to cardiac hypertrophy induced by transverse aortic constriction with prominent cardiac fibrosis, particularly when compared with the levels in unstressed cardiomyocytes. In n...

2012
Ian M C Dixon

We are pleased to introduce a new thematic series dealing with cardiac fibrosis and its association with cardiovascular diseases. A wide variety of cardiovascular diseases are associated with cardiac fibrosis, which is now widely recognized to be not a secondary, but rather a primary contributor to cardiac dysfunction. The purpose of the current series of papers and reviews is to provide the re...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2015
Havovi Chichger Alexander Vang Kelly A O'Connell Peng Zhang Ulrike Mende Elizabeth O Harrington Gaurav Choudhary

Pulmonary hypertension (PH) eventually leads to right ventricular (RV) fibrosis and dysfunction that is associated with increased morbidity and mortality. Although angiotensin II plays an important role in RV remodeling associated with hypoxic PH, the molecular mechanisms underlying RV fibrosis in PH largely remain unresolved. We hypothesized that PKC-p38 signaling is involved in RV collagen ac...

Journal: :American journal of physiology. Heart and circulatory physiology 2015
Kiyoshi Yamagami Toru Oka Qi Wang Takamaru Ishizu Jong-Kook Lee Keiko Miwa Hiroshi Akazawa Atsuhiko T Naito Yasushi Sakata Issei Komuro

Although cardiac fibrosis causes heart failure, its molecular mechanisms remain elusive. In this study, we investigated the mechanisms of cardiac fibrosis and examined the effects of the antifibrotic drug pirfenidone (PFD) on chronic heart failure. To understand the responsible mechanisms, we generated an in vivo pressure-overloaded heart failure model via transverse aortic constriction (TAC) a...

2016
Lichan Tao Yihua Bei Ping Chen Zhiyong Lei Siyi Fu Haifeng Zhang Jiahong Xu Lin Che Xiongwen Chen Joost PG Sluijter Saumya Das Dragos Cretoiu Bin Xu Jiuchang Zhong Junjie Xiao Xinli Li

Dysregulation of microRNAs has been implicated in many cardiovascular diseases including fibrosis. Here we report that miR-433 was consistently elevated in three models of heart disease with prominent cardiac fibrosis, and was enriched in fibroblasts compared to cardiomyocytes. Forced expression of miR-433 in neonatal rat cardiac fibroblasts increased proliferation and their differentiation int...

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