نتایج جستجو برای: amyloid beta aβ

تعداد نتایج: 218049  

Journal: :Advanced nanoBiomed research 2023

Fluorescence imaging of amyloid beta (Aβ) polypeptide using a small-molecule fluorophore is practical system for Alzheimer's disease (AD) diagnosis in clinical and research fields. Herein, molecular designing strategy new fluorogenic probe that equips bathochromic emission shift enhanced binding affinity toward Aβ fibril through structural optimization suggested. Thioflavin T (ThT)-derived fluo...

Different treatment strategies of Alzheimer's disease (AD) are being studied for treating or slowing the progression of AD. Many pharmaceutically important regulation systems operate through proteins as drug targets. Here, we investigate the drug target proteins in beta-amyloid (Aβ) injected rat hippocampus treated with Lavandula angustifolia (LA) by proteomics techniques. The reported study sh...

Interactions of 3,3',4,4'-tetrahydroxybiphenyl (BPT) and three isomeric 3,3",4,4"-tetrahydroxyterphenyls (OTT, MTT, PTT) with Alzheimer’s amyloid-β peptide (Aβ) were studied by molecular dynamics simulation and molecular docking. Structural parameters such as Root-mean-square derivations (RMSD), radial distribution function (RDF), helix percentage and other physical parameters were obtained. Th...

Banafshe Esmailzade, Iraj Sohrabi, Maliheh Nobakht, Nahid Rahbar Rooshandel, Pejman Mortazavi, Seyed Mohammad Hoseini, Shila Omidzahir,

Background: Alzheimer’s disease (AD) is a neurodegenerative disorder with progressive loss of cognitive abilities and memory loss. The aim of this study was to compare neuropathological changes in hippocampus and brain cortex in a rat model of AD. Methods: Adult male Albino Wistar rats (weighing 250-300 g) were used for behavioral and histopathological studies. The rats were randomly assigned t...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Gunjan Joshi Youjian Chi Zheping Huang Yanzhuang Wang

Golgi fragmentation occurs in neurons of patients with Alzheimer's disease (AD), but the underlying molecular mechanism causing the defects and the subsequent effects on disease development remain unknown. In this study, we examined the Golgi structure in APPswe/PS1E9 transgenic mouse and tissue culture models. Our results show that accumulation of amyloid beta peptides (Aβ) leads to Golgi frag...

2015
Jacopo C. DiFrancesco Martina Longoni Fabrizio Piazza

Amyloid-related imaging abnormalities (ARIA) represent the major severe side effect of amyloid-beta (Aβ) immunotherapy for Alzheimer's disease (AD). Early biomarkers of ARIA represent an important challenge to ensure safe and beneficial effects of immunotherapies, given that different promising clinical trials in prodromal and subjects at risk for AD are underway. The recent demonstration that ...

Journal: :Alzheimers & Dementia 2023

Background Down syndrome (DS) is characterized by earlier beta-amyloid (Aβ) plaque accumulation and an increased prevalence of Alzheimer’s disease (AD) due to trisomy 21 triplication the amyloid precursor protein (APP) gene. While age AD onset in DS, pathology progression non-DS populations follows similar trends DS cohort. This study compares rate Aβ between measured with [C-11]PiB at same sit...

2012
Hitomi Ohta Shigeyuki Arai Kenji Akita Tsunetaka Ohta Shigeharu Fukuda

Beta-amyloid (Aβ) peptides are considered to play a major role in the pathogenesis of Alzheimer's disease (AD) and molecules that can prevent pathways of Aβ toxicity may be potential therapeutic agents for treatment of AD. We have previously reported that NK-4, a cyanine photosensitizing dye, displays neurotrophic and antioxidant activities. In this study, we report the effects of NK-4 on the t...

Journal: :Neurobiology of aging 2014
Sterling C Johnson Bradley T Christian Ozioma C Okonkwo Jennifer M Oh Sandra Harding Guofan Xu Ansel T Hillmer Dustin W Wooten Dhanabalan Murali Todd E Barnhart Lance T Hall Annie M Racine William E Klunk Chester A Mathis Barbara B Bendlin Catherine L Gallagher Cynthia M Carlsson Howard A Rowley Bruce P Hermann N Maritza Dowling Sanjay Asthana Mark A Sager

To determine the relationship between amyloid burden and neural function in healthy adults at risk for Alzheimer's Disease (AD), we used multimodal imaging with [C-11]Pittsburgh compound B positron emission tomography, [F-18]fluorodeoxyglucose, positron emission tomography , and magnetic resonance imaging, together with cognitive measurement in 201 subjects (mean age, 60.1 years; range, 46-73 y...

2013
Ayman ElAli Serge Rivest

Alzheimer's disease (AD) is a progressive neurodegenerative disorder that affects elderly persons, evolving with age to reach severe cognitive impairment. Amyloid deposits and neurofibrillary tangles constitute the main pathological hallmarks of AD. Amyloid deposits are initiated by the excessive production and accumulation of beta-amyloid (Aβ) peptides in the brain. The dysfunction of the Neur...

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