نتایج جستجو برای: 1 antitrypsin a1at

تعداد نتایج: 2753541  

2012
Michael Sjoding Kyle Hogarth

Alpha one antitrypsin protein (A1AT), is encoded on the SERPINA1 (serpin peptidase inhibitor, claude A, member 1 gene), (OMIM 107400), located on chromosome 14q32.1 and functions as inhibitor of the enzyme neutrophil elastase. People with a low serum level of this protein are described as having the alpha-1-antrypsin deficiency (A1ATD), (OMIM #613490), one of the most common heritable disorders...

Journal: :Clinical chemistry 2011
Cory E Bystrom

1-Antitrypsin (A1AT) 2 deficiency is a serious, lifethreatening disorder driven by abnormally low circulating concentrations of A1AT, which lead to protease– antiprotease imbalance. This imbalance accentuates protease activity and can lead to tissue damage, the most important of which leads to impairment of lung function (1 ). The apparent simplicity of diagnosing this disorder by a single meas...

2005
Peter C. Harpel

An enzyme-linked immunosorbent assay (ELISA) has been developed for the quantification of cr,-antitrypsin-human leukocyte elastase (a,AT-E) complexes. In the ELISA, the a,AT-E complex is bound to a surface by rabbit antileukocyte elastase antibody, and the inhibitor-proteinase cornplex is quantified by a second antibody, rabbit anti-a1antitrypsin F(ab’)2. labeled with alkaline phosphatase. a1AT...

2016
Te-Yao Hsu Hao Lin Hsuan-Ning Hung Kuender D. Yang Chia-Yu Ou Ching-Chang Tsai Hsin-Hsin Cheng Su-Hai Chung Bi-Hua Cheng Yi-Hsun Wong An Kuo Chou Chang-Chun Hsiao Daniela Flavia Hozbor

BACKGROUND Edwards syndrome (ES) is a severe chromosomal abnormality with a prevalence of about 0.8 in 10,000 infants born alive. The aims of this study were to identify candidate proteins associated with ES pregnancies from amniotic fluid supernatant (AFS) using proteomics, and to explore the role of biological networks in the pathophysiology of ES. METHODS AFS from six second trimester preg...

ژورنال: :hepatitis monthly 0
ignacio blanco the principality of asturias biomedical research office (oib-ficyt), oviedo, spain and alpha1-antitrypsin deficiency spanish registry, spain +34-985252481, [email protected]; uluhqsmoo}yu~yt{iofdew}w{mosyb{mom{ka~ewmsus{ch office (oib-ficyt), oviedo, spain and alpha1-antitrypsin deficiency spanish registry, spain +34-985252481, [email protected]; the principality of asturias biomedical research office (oib-ficyt), oviedo, spain and alpha1-antitrypsin deficiency spanish registry, spain +34-985252481, [email protected] frederick j. de serres national institute of environmental health sciences, research triangle park, nc, usa, usa victoriano cárcaba department of internal medicine, ‘valle del nalón hospital, principado de asturias, spain, spain beatríz lara hospital universitario arnau de vilanova, avda, institut de recerca biomédica de lleida (irb), ciberes instituto salud carlos iii, pneumology service, institut de recerca hospital universitari vall d’hebron, spain enrique fernández-bustillo biostatistics unit, central university hospital of asturias, spain

background currently, there is a remarkable lack of genetic epidemiological studies on alpha 1-antitrypsin (aat) deficiency in about half of the 193 countries of the world. this fact impedes the establishment of a true prevalence pattern of this deleterious hereditary disorder in extensive regions of human population. objectives the aim of the present study was to generate detailed maps of the ...

Journal: :Postgraduate medicine 2017
John A Bilello Forest S Tennant

OBJECTIVE To use biomarkers to gain insight into and gauge the residual (post-treatment) level of inflammation in two groups of intensively treated patients with severe chronic pain. METHODS Three study groups were analyzed, and included: (i) patients (n = 90) with chronic intractable pain (CIP), (ii) patients (n = 26) with chronic pain and MRI-documented arachnoiditis (ARC) and (iii) normal ...

Journal: :Clinical chemistry 2009
Denis Roche Alexandra Mesner Malik Al Nakib Frederic Leonard Philippe Beaune

BACKGROUND Alpha1-Antitrypsin (A1AT) deficiency is currently detectable by protein immunoassay, phenotyping, and genotyping of the S and Z mutations, but no fully automated method for standard biochemical analyzers is yet available. Here, we present a method that measures the antitryptic activity in serum. This method is rapid, automated, and allows the easy evaluation of a large cohort of pati...

2017
Donald R Royall Safa Al-Rubaye Ram Bishnoi Raymond F Palmer

The latent variable "δ" (for "dementia") uniquely explains dementia severity. Depressive symptoms are independent predictors of δ. We explored 115 serum proteins as potential causal mediators of the effect of depressive symptoms on δ in a large, ethnically diverse, longitudinal cohort. All models were adjusted for age, apolipoprotein E, education, ethnicity, gender, hemoglobin A1c, and homocyst...

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