نتایج جستجو برای: ژن pik3ca

تعداد نتایج: 18195  

2013
Hironobu Shigaki Yoshifumi Baba Masayuki Watanabe Asuka Murata Takatsugu Ishimoto Masaaki Iwatsuki Shiro Iwagami Katsuhiko Nosho Hideo Baba

Purpose: PIK3CA encodes the catalytic subunit of PI3K, p110a. Mutant PIK3CA stimulates the AKT pathway and promotes cancer cell proliferation. PIK3CA mutations have been associated with poor prognosis in patientswith colorectal or lung cancer. In contrast, the relationship between PIK3CAmutations and favorable prognoses has been shown in breast cancer. However, the influence of PIK3CAmutations ...

Journal: :Cancer research 2004
Ian G Campbell Sarah E Russell David Y H Choong Karen G Montgomery Marianne L Ciavarella Christine S F Hooi Briony E Cristiano Richard B Pearson Wayne A Phillips

Phosphatidylinositol 3'-kinases are lipid kinases with important roles in neoplasia. Recently, a very high frequency of somatic mutations in PIK3CA has been reported among a large series of colorectal cancers. However, the relevance of PIK3CA mutation in other cancer types remains unclear because of the limited number of tumors investigated. We have screened a total of 284 primary human tumors ...

Journal: :Cancer research 2007
Xiao-Ning Guo Ashwani Rajput Rebecca Rose Jennie Hauser Alexander Beko Karen Kuropatwinski Charles LeVea Robert M Hoffman Michael G Brattain Jing Wang

Mutations in the PIK3CA gene are common in human cancers, including colon cancer. We compared two pairs of colon cancer cells (HCT116 and DLD1) bearing only the wild-type (WT) or mutant (MUT) PIK3CA allele for their survival capacity under stress conditions in vitro as well as their metastatic properties in an in vivo orthotopic model. When subjected to growth factor deprivation stress (GFDS), ...

2016
Yujun Hao Yardena Samuels Qingling Li Dawid Krokowski Bo-Jhih Guan Chao Wang Zhicheng Jin Bohan Dong Bo Cao Xiujing Feng Min Xiang Claire Xu Stephen Fink Neal J. Meropol Yan Xu Ronald A. Conlon Sanford Markowitz Kenneth W. Kinzler Victor E. Velculescu Henri Brunengraber Joseph E. Willis Thomas LaFramboise Maria Hatzoglou Guo-Fang Zhang Bert Vogelstein Zhenghe Wang

Cancer cells often require glutamine for growth, thereby distinguishing them from most normal cells. Here we show that PIK3CA mutations reprogram glutamine metabolism by upregulating glutamate pyruvate transaminase 2 (GPT2) in colorectal cancer (CRC) cells, making them more dependent on glutamine. Compared with isogenic wild-type (WT) cells, PIK3CA mutant CRCs convert substantially more glutami...

2014
Gieri Cathomas

PIK3CA, the catalytic subunit of PI3K, is mutated in many different tumors, including colorectal cancer (CRC). Mutations of PIK3CA have been reported in 10-20% of CRC, about 80% of mutations found in two hot spots in exon 9 and exon 20. In RAS wild-type CRC, PIK3CA mutations have been associated with a worse clinical outcome and with a negative prediction of a response to targeted therapy by an...

2012
Xiaoyun Liao Teppei Morikawa Paul Lochhead Yu Imamura Aya Kuchiba Mai Yamauchi Katsuhiko Nosho Zhi Rong Qian Reiko Nishihara Jeffrey A. Meyerhardt Charles S. Fuchs Shuji Ogino

Purpose:Mutations in PIK3CA [the gene encoding the p110a catalytic subunit of phosphatidylinositide3-kinase (PI3K)] play an important role in colorectal carcinogenesis. Experimental evidence suggests that PIK3CA exon 9 and exon 20mutations trigger different biologic effects, and that concomitant mutations in both exons 9 and 20 synergistically enhance tumorigenic effects. Thus, we hypothesized ...

2011
Jessica R. Adams Keli Xu Jeff C. Liu Natalia M. Ruiz Agamez Amanda J. Loch Ruth G. Wong Wei Wang Katherine L. Wright Timothy F. Lane Eldad Zacksenhaus Sean E. Egan

PIK3CA, which codes for the p110a catalytic subunit of phosphatidylinositol 3-kinase, is one of the most frequently mutated genes in human breast cancer. Here, we describe a mouse model for PIK3CA-induced breast cancer by using the ROSA26 (R26) knock-in system, in which targeted Pik3ca alleles can be activated through transgenic expression of Cre recombinase. We mated Pik3ca and Pik3ca knock-in...

2017
Takashi Takeshita Yutaka Yamamoto Mutsuko Yamamoto-Ibusuki Mai Tomiguchi Aiko Sueta Keiichi Murakami Yoko Omoto Hirotaka Iwase

BACKGROUND The measurement of ESR1 and PIK3CA mutations in plasma cell-free DNA (cfDNA) has been studied as a non-invasive method to quickly assess and monitor endocrine therapy (ET) resistant metastatic breast cancer (MBC) patients. METHODS The subjects of this retrospective study were a total of 185 plasma samples from 86 estrogen receptor-positive BC patients, of which 151 plasma samples w...

Journal: :Cancer research 2005
Katsutoshi Oda David Stokoe Yuji Taketani Frank McCormick

The phosphatidylinositol 3'-kinase (PI3K) pathway is activated in many human cancers. In addition to inactivation of the PTEN tumor suppressor gene, mutations or amplifications of the catalytic subunit alpha of PI3K (PIK3CA) have been reported. However, the coexistence of mutations in these two genes seems exceedingly rare. As PTEN mutations occur at high frequency in endometrial carcinoma, we ...

Journal: :Cancer research 2008
Katherine Stemke-Hale Ana Maria Gonzalez-Angulo Ana Lluch Richard M Neve Wen-Lin Kuo Michael Davies Mark Carey Zhi Hu Yinghui Guan Aysegul Sahin W Fraser Symmans Lajos Pusztai Laura K Nolden Hugo Horlings Katrien Berns Mien-Chie Hung Marc J van de Vijver Vicente Valero Joe W Gray René Bernards Gordon B Mills Bryan T Hennessy

Phosphatidylinositol 3-kinase (PI3K)/AKT pathway aberrations are common in cancer. By applying mass spectroscopy-based sequencing and reverse-phase protein arrays to 547 human breast cancers and 41 cell lines, we determined the subtype specificity and signaling effects of PIK3CA, AKT, and PTEN mutations and the effects of PIK3CA mutations on responsiveness to PI3K inhibition in vitro and on out...

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