نتایج جستجو برای: ژن نوکلئوفسمین npm1

تعداد نتایج: 16927  

Journal: :Haematologica 2008
Arcangelo Liso Filippo Castiglione Antonio Cappuccio Fabrizio Stracci Richard F Schlenk Sergio Amadori Christian Thiede Susanne Schnittger Peter J M Valk Konstanze Döhner Massimo F Martelli Markus Schaich Jürgen Krauter Arnold Ganser Maria P Martelli Niccolò Bolli Bob Löwenberg Torsten Haferlach Gerhard Ehninger Franco Mandelli Hartmut Döhner Franziska Michor Brunangelo Falini

Acute myeloid leukemia with mutated NPM1 gene and aberrant cytoplasmic expression of nucleophosmin (NPMc(+) acute myeloid leukemia) shows distinctive biological and clinical features. Experimental evidence of the oncogenic potential of the nucleophosmin mutant is, however, still lacking, and it is unclear whether other genetic lesion(s), e.g. FLT3 internal tandem duplication, cooperate with NPM...

2008
Arcangelo Liso Filippo Castiglione Antonio Cappuccio Fabrizio Stracci Richard F. Schlenk Sergio Amadori Christian Thiede Susanne Schnittger Peter J.M. Valk Konstanze Döhner Massimo F. Martelli Markus Schaich Jürgen Krauter Arnold Ganser Maria P. Martelli Niccolò Bolli Bob Löwenberg Torsten Haferlach Gerhard Ehninger Franco Mandelli Hartmut Döhner Franziska Michor Brunangelo Falini

Acute myeloid leukemia with mutated NPM1 gene and aberrant cytoplasmic expression of nucleophosmin (NPMc acute myeloid leukemia) shows distinctive biological and clinical features. Experimental evidence of the oncogenic potential of the nucleophosmin mutant is, however, still lacking, and it is unclear whether other genetic lesion(s), e.g. FLT3 internal tandem duplication, cooperate with NPM1 m...

Journal: :Asian Pacific journal of cancer prevention : APJCP 2011
Ekchol Mukda Katsarin Pintaraks Rachchadol Sawangpanich Surapon Wiangnon Samart Pakakasama

Mutations of receptor tyrosine kinases are implicated in the constitutive activation and development of human hematologic malignancies. Mutations in fms-like tyrosine kinase 3 (FLT3) gene including internal tandem duplication (ITD) and point mutation in the tyrosine kinase domain (TKD) as well as in nucleoplasmin (NPM1) gene are associated with pathogenesis of acute myeloblastic leukemia (AML)....

2016
Shuaishuai Zhang Fengxian Qin Liyuan Yang Jingrong Xian Qin Zou Hongjun Jin Lu Wang Ling Zhang

Nucleophosmin (NPM1) - a gene that encodes for a nuclear protein with multiple functions. Mutations in NPM1 are seen in approximately one-third of acute myeloid leukemia (AML) and are generally associated with good response to induction chemotherapy. However, the mechanisms underlying this chemosensitivity are still unknown. Recent studies have established that nuclear factor-κB (NF-κB) activat...

2016
Vibeke Andresen Bjarte S Erikstein Herschel Mukherjee André Sulen Mihaela Popa Steinar Sørnes Håkon Reikvam Kok-Ping Chan Randi Hovland Emmet McCormack Øystein Bruserud Andrew G Myers Bjørn T Gjertsen

Mutated nucleophosmin 1 (NPM1) acts as a proto-oncogene and is present in ~30% of patients with acute myeloid leukemia (AML). Here we examined the in vitro and in vivo anti-leukemic activity of the NPM1 and chromosome region maintenance 1 homolog (CRM1) interacting natural product avrainvillamide (AVA) and a fully syntetic AVA analog. The NPM1-mutated cell line OCI-AML3 and normal karyotype pri...

2017
Jo-Anne van der Krogt Marlies Vanden Bempt Julio Finalet Ferreiro Nicole Mentens Kris Jacobs Ursula Pluys Kathleen Doms Ellen Geerdens Anne Uyttebroeck Pascal Pierre Lucienne Michaux Timothy Devos Peter Vandenberghe Thomas Tousseyn Jan Cools Iwona Wlodarska

Anaplastic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma is characterized by 2p23/ALK aberrations, including the classic t(2;5)(p23;q35)/NPM1-ALK rearrangement present in ~80% of cases and several variant t(2p23/ALK) occurring in the remaining cases. The ALK fusion partners play a key role in the constitutive activation of the chimeric protein and its subcellular localization. U...

Journal: :Haematologica 2013
Florent Dumézy Aline Renneville Caroline Mayeur-Rousse Olivier Nibourel Elise Labis Claude Preudhomme

AML with translocation t(3;5) belongs to the “AML with myelodysplasia-related changes” defined in the 2008 WHO classification. The incidence of this balanced abnormality is less than 0.5% of AML. The identified breakpoints occur at 3q25.1 on chromosome 3 and at 5q34 on chromosome 5, where the nucleolar phosphoprotein nucleophosmin 1 (NPM1) is located. At locus 3q25.1, Yoneda-Kato et al. identif...

Journal: :Haematologica 2011
Maria Stefania De Propris Sara Raponi Daniela Diverio Maria Laura Milani Giovanna Meloni Brunangelo Falini Robin Foà Anna Guarini

The CD33 antigen is expressed on the blast cells of most cases of acute myeloid leukemia and represents a suitable tumor-associated target antigen for antibody-based therapies. The aim of this study was to investigate the relationship between the CD33 levels quantified by mean fluorescence intensity and antibody binding capacity, and the presence/absence of NPM1 and FLT3 gene mutations in 99 ne...

2016
Min Huang Jacqueline S. Garcia Daniel Thomas Li Zhu Le Xuan Truong Nguyen Steven M. Chan Ravindra Majeti Bruno C. Medeiros Beverly S. Mitchell

The mechanisms underlying activation of the BET pathway in AML cells remain poorly understood. We have discovered that autophagy is activated in acute leukemia cells expressing mutant nucleophosmin 1 (NPMc+) or MLL-fusion proteins. Autophagy activation results in the degradation of NPM1 and HEXIM1, two negative regulators of BET pathway activation. Inhibition of autophagy with pharmacologic inh...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Diana M Mitrea Christy R Grace Marija Buljan Mi-Kyung Yun Nicholas J Pytel John Satumba Amanda Nourse Cheon-Gil Park M Madan Babu Stephen W White Richard W Kriwacki

Nucleophosmin (NPM1) is a multifunctional phospho-protein with critical roles in ribosome biogenesis, tumor suppression, and nucleolar stress response. Here we show that the N-terminal oligomerization domain of NPM1 (Npm-N) exhibits structural polymorphism by populating conformational states ranging from a highly ordered, folded pentamer to a highly disordered monomer. The monomer-pentamer equi...

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