نتایج جستجو برای: تست mpn

تعداد نتایج: 18267  

Journal: :Blood 2010
Sylvain Thepot Raphael Itzykson Valerie Seegers Emmanuel Raffoux Bruno Quesnel Yasmine Chait Lucile Sorin Francois Dreyfus Thomas Cluzeau Jacques Delaunay Laurence Sanhes Virginie Eclache Caroline Dartigeas Pascal Turlure Stephanie Harel Celia Salanoubat Jean-Jacques Kiladjian Pierre Fenaux Lionel Adès

Transformation of Philadelphia (Ph)-negative myeloproliferative neoplasms (MPNs) to myelodysplastic syndrome (MDS) or acute myeloid leukemia (AML) is associated with poor response to chemotherapy and short survival. Fifty-four patients with Ph-negative MPN (including 21 essential thrombocythemia [ET], 21 polycythemia vera [PV], 7 primary myelofibrosis, and 5 unclassified MPN) who had progressed...

2015
Bruno A. Cardoso Hélio Belo João T. Barata António M. Almeida Chih-Hsin Tang

The classical BCR-ABL-negative Myeloproliferative Neoplasms (MPN) are a group of heterogeneous haematological diseases characterized by constitutive JAK-STAT pathway activation. Targeted therapy with Ruxolitinib, a JAK1/2-specific inhibitor, achieves symptomatic improvement but does not eliminate the neoplastic clone. Similar effects are seen with histone deacetylase inhibitors (HDACi), albeit ...

2016
Takafumi Shimizu Lucia Kubovcakova Ronny Nienhold Jakub Zmajkovic Sara C Meyer Hui Hao-Shen Florian Geier Stephan Dirnhofer Paola Guglielmelli Alessandro M Vannucchi Jelena D Milosevic Feenstra Robert Kralovics Stuart H Orkin Radek C Skoda

Myeloproliferative neoplasm (MPN) patients frequently show co-occurrence of JAK2-V617F and mutations in epigenetic regulator genes, including EZH2 In this study, we show that JAK2-V617F and loss of Ezh2 in hematopoietic cells contribute synergistically to the development of MPN. The MPN phenotype induced by JAK2-V617F was accentuated in JAK2-V617F;Ezh2(-/-) mice, resulting in very high platelet...

2013
Asunción Martínez Laure-Anne Ventouras Samuel T. Wilson David M. Karl Edward F. DeLong

Aerobic degradation of methylphosphonate (MPn) by marine bacterioplankton has been hypothesized to contribute significantly to the ocean's methane supersaturation, yet little is known about MPn utilization by marine microbes. To identify the microbial taxa and metabolic functions associated with MPn-driven methane production we performed parallel metagenomic, metatranscriptomic, and functional ...

Journal: :Applied and environmental microbiology 1999
T R Slifko D E Huffman J B Rose

Cryptosporidium is globally established as a contaminant of drinking and recreational waters. A previously described cell culture infectivity assay capable of detecting infectious oocysts was adapted to quantify viable oocysts through sporozoite invasion and clustering of foci. Eight experiments were performed by using oocysts less than 4 months of age to inoculate host HCT-8 cell monolayers. O...

2016
Susumu S. Kobayashi Shireen Vali Ansu Kumar Neeraj Singh Taher Abbasi Peter P. Sayeski

Previous studies have shown that the bone marrow micro-environment supports the myeloproliferative neoplasms (MPN) phenotype including via the production of cytokines that can induce resistance to frontline MPN therapies. However, the mechanisms by which this occurs are poorly understood. Moreover, the ability to rapidly identify drug agents that can act as adjuvants to existing MPN frontline t...

2018
Sarah Hintermair Elisabeth Zwickl-Traxler Martin Pecherstorfer Josef Singer

Myeloproliferative neoplasms (MPN), classified as polycythemia vera (PV), essential thrombocytosis (ET) and myelofibrosis (MF) are stem-cell derived disorders. Mutations in either the januskinase-2 (JAK-2) or the calreticulin (CALR) gene are characteristic for MPN and may result in enhanced proliferation of red blood cells, white blood cells and platelets, and thus increase the risk for vascula...

Journal: :Blood 2011
Shubha Anand Frances Stedham Emma Gudgin Peter Campbell Philip Beer Anthony R Green Brian J P Huntly

Myeloproliferative neoplasms (MPNs) are associated with recurrent activating mutations of signaling proteins such as Janus kinase 2 (JAK2). However, the actual downstream signaling events and how these alter myeloid homeostasis are poorly understood. We developed an assay to measure basal levels of phosphorylated signaling intermediates by flow cytometry during myeloid differentiation in MPN pa...

2017
Nana Brochmann Esben Meulengracht Flachs Anne Illemann Christensen Christen Lykkegaard Andersen Knud Juel Hans Carl Hasselbalch Ann-Dorthe Zwisler

OBJECTIVE The Department of Hematology, Zealand University Hospital, Denmark, and the National Institute of Public Health, University of Southern Denmark, created the first nationwide, population-based, and the most comprehensive cross-sectional health-related quality of life (HRQoL) survey of patients with myeloproliferative neoplasms (MPNs). In Denmark, all MPN patients are treated in public ...

Journal: :Blood 2015
Michael R Savona Luca Malcovati Rami Komrokji Ramon V Tiu Tariq I Mughal Attilio Orazi Jean-Jacques Kiladjian Eric Padron Eric Solary Raoul Tibes Raphael Itzykson Mario Cazzola Ruben Mesa Jaroslaw Maciejewski Pierre Fenaux Guillermo Garcia-Manero Aaron Gerds Guillermo Sanz Charlotte M Niemeyer Francisco Cervantes Ulrich Germing Nicholas C P Cross Alan F List

Myelodysplastic syndromes (MDS) and myeloproliferative neoplasms (MPN) are hematologically diverse stem cell malignancies sharing phenotypic features of both myelodysplastic syndromes and myeloproliferative neoplasms. There are currently no standard treatment recommendations for most adult patients with MDS/MPN. To optimize efforts to improve the management and disease outcomes, it is essential...

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