نتایج جستجو برای: wilms tumor

تعداد نتایج: 428548  

Journal: :Sao Paulo medical journal = Revista paulista de medicina 2000
R Defavery J A Lemos S Kashima J E Bernardes C A Scridelli D T Covas L G Tone

CONTEXT Mutations of the p53 tumor suppressor gene are the most frequent alterations observed in human neoplasias affecting adults. In pediatric oncology, however, they have seldom been identified. Wilms' tumor is a renal neoplasia commonly occurring in children and is associated with mutations of the WT1 gene. The correlation between Wilms' tumor and alterations of the p53 gene has not been we...

Journal: :American Journal of Roentgenology 1971

Journal: :CA: A Cancer Journal for Clinicians 1982

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2011

2010
Muhammad A Khattak Hilary L Martin Ganessan Kichenadasse

Wilms' tumor is one of the most common pediatric malignancies. Survival rates have increased dramatically over the last few decades. This increased survival means that there is an ever growing population of patients susceptible to the late effects of their initial therapy. Survivors of Wilms' tumor have a substantially higher rate of development of secondary neoplasms compared to general popula...

2006
Yu Rong Long Cheng Hongxiu Ning Jizhen Zou Yuanjiang Zhang Fang Xu Li Liu Zhijie Chang Xin-Yuan Fu

Transcription factor Wilms’ tumor 1 (WT1) was originally identified as a tumor suppressor for Wilms’ tumor, but it is also overexpressed in a variety of cancer cells, suggesting a potential oncogenic function of WT1. It is important to understand molecular mechanisms underlying these dual functions of WT1 in tumorigenesis. In the current study, we report a synergistic role for signal transducer...

Journal: :Cancer research 1991
Y Kaneko C Homma N Maseki M Sakurai J Hata

Chromosomes and histology were successfully studied in 33 childhood renal tumors. Thirty-one tumors were classified as one of four subtypes of Wilms' tumor. Of 24 typical Wilms' tumors, 12 had hyperdiploidy with nonrandom trisomies, mostly including +6 and/or +12. Three typical Wilms' tumors with an 11p13 deletion or a pericentric inversion with a break in 11p13 were not associated with aniridi...

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