نتایج جستجو برای: neuronal injury

تعداد نتایج: 427898  

Journal: :Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 2010

Journal: :Journal of Cerebral Blood Flow & Metabolism 1992

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
Yongfu Wang Ji-Hoon Song Janna V Denisova Won-Mee Park Joseph D Fontes Andrei B Belousov

In the mammalian CNS, excessive release of glutamate and overactivation of glutamate receptors are responsible for the secondary (delayed) neuronal death following neuronal injury, including ischemia, traumatic brain injury (TBI), and epilepsy. The coupling of neurons by gap junctions (electrical synapses) increases during neuronal injury. We report here that the ischemic increase in neuronal g...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
John E Greer Melissa J McGinn John T Povlishock

Traumatic axonal injury (TAI) is a consistent component of traumatic brain injury (TBI) and is associated with much of its morbidity. Little is known regarding the long-term retrograde neuronal consequences of TAI and/or the potential that TAI could lead to anterograde axonal reorganization and repair. To investigate the repertoire of anterograde and retrograde responses triggered by TIA, Thy1-...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2009
Dorothy P Schafer Smita Jha Fudong Liu Trupti Akella Louise D McCullough Matthew N Rasband

Many factors contribute to nervous system dysfunction and failure to regenerate after injury or disease. Here, we describe a previously unrecognized mechanism for nervous system injury. We show that neuronal injury causes rapid, irreversible, and preferential proteolysis of the axon initial segment (AIS) cytoskeleton independently of cell death or axon degeneration, leading to loss of both ion ...

Ali Gorji, Ali Jahanbazi Jahan-Abad, Hassan Hosseini Ravandi, Maryam Khaleghi Ghadiri, Sajad Sahab Negah, Sedigheh Ghasemi, Walter Stummer,

Traumatic brain injury (TBI) is a disruption in the brain functions following a head trauma. Cell therapy may provide a promising treatment for TBI. Human neural stem cells cultured in self-assembling peptide scaffolds have been proposed as a potential novel method for cell replacement treatment after TBI. In the present study, we accessed the effects of human neural stem/progenitor cells (hNS/...

Objective(s): Paeoniflorin (PF) has anti-oxidation, anti-inflammation, anti-apoptosis, and neuroprotection pharmacological effects against ischemic injury. The aim of the present study was to investigate the neuroprotection mechanisms of PF in cerebral ischemia-reperfusion injury rats.Materials and Methods: We established an animal model of cerebral infarct by occlusion of the middle cerebral a...

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