نتایج جستجو برای: myo

تعداد نتایج: 3424  

Journal: :European journal of endocrinology 2012
Gabriella Donà Chiara Sabbadin Cristina Fiore Marcantonio Bragadin Francesco L Giorgino Eugenio Ragazzi Giulio Clari Luciana Bordin Decio Armanini

OBJECTIVE Possibly due to a deficiency of insulin mediators, polycystic ovary syndrome (PCOS) is often associated with insulin resistance (IR) and hyperinsulinemia, likely responsible for an elevated production of reactive oxygen species. We investigated oxidative-related alterations in erythrocytes and anti-inflammatory effects of inositol in women with PCOS before and after treatment with myo...

Journal: :Cancer prevention research 2016
Stephen Lam Sumithra J Mandrekar Yaron Gesthalter Katie L Allen Ziegler Drew K Seisler David E Midthun Jenny T Mao Marie Christine Aubry Annette McWilliams Don D Sin Tawimas Shaipanich Gang Liu Evan Johnson Andrea Bild Marc E Lenburg Diana N Ionescu John Mayo Joanne Eunhee Yi Henry Tazelaar William S Harmsen Judith Smith Avrum E Spira Jennifer Beane Paul J Limburg Eva Szabo

Previous preclinical studies and a phase I clinical trial suggested that myo-inositol may be a safe and effective lung cancer chemopreventive agent. We conducted a randomized, double blind, placebo-controlled phase IIb study to determine the chemopreventive effects of myo-inositol in smokers with bronchial dysplasia. Smokers with ≥1 site of dysplasia identified by autofluorescence bronchoscopy-...

Journal: :Journal of bacteriology 2006
Nuno Borges Luís G Gonçalves Marta V Rodrigues Filipa Siopa Rita Ventura Christopher Maycock Pedro Lamosa Helena Santos

Archaeoglobus fulgidus accumulates di-myo-inositol phosphate (DIP) and diglycerol phosphate (DGP) in response to heat and osmotic stresses, respectively, and the level of glycero-phospho-myo-inositol (GPI) increases primarily when the two stresses are combined. In this work, the pathways for the biosynthesis of these three compatible solutes were established based on the detection of the releva...

Journal: :Current Biology 2012
Pralay Majumder George Aranjuez Joseph Amick Jocelyn A. McDonald

BACKGROUND Localized actomyosin contraction couples with actin polymerization and cell-matrix adhesion to regulate cell protrusions and retract trailing edges of migrating cells. Although many cells migrate in collective groups during tissue morphogenesis, mechanisms that coordinate actomyosin dynamics in collective cell migration are poorly understood. Migration of Drosophila border cells, a g...

Journal: :The Journal of biological chemistry 1982
V Prpić P F Blackmore J H Exton

The uptake of myo-inositol into isolated rat liver parenchymal cells and its incorporation into phosphatidylinositol were studied by isotopic and chemical means. The uptake was concentrationand time-dependent, but a maximal rate could not be defined. The intracellular concentration of myo-inositol never exceeded the extracellular concentration, and the uptake was not affected by inhibitors of m...

Journal: :The Biochemical journal 1988
L Stephens P T Hawkins N Carter S B Chahwala A J Morris A D Whetton P C Downes

When myo-[3H]inositol-prelabelled primary-cultured murine bone-marrow-derived macrophages were challenged with platelet-activating factor (PAF; 200 ng/ml), there was a rapid (2.5-fold at 10 s) rise in the intracellular concentration of D-myo-[3H]inositol 1,4,5-trisphosphate, followed by a rise in myo-[3H]inositol tetrakisphosphate. myo-[3H]Inositol tetrakisphosphate fractions were isolated by h...

Journal: :American journal of physiology. Regulatory, integrative and comparative physiology 2004
Georg Schlieper Jie-Hoon Kim Andrei Molojavyi Christoph Jacoby Tim Laussmann Ulrich Flögel Axel Gödecke Jürgen Schrader

Myoglobin knockout (myo-/-) mice were previously reported to show no obvious phenotype but revealed several compensatory mechanisms that include increases in cardiac capillary density, coronary flow, and hemoglobin. The aim of this study was to investigate whether severe hypoxic stress can exhaust these compensatory mechanisms and whether this can be monitored on the gene and protein level. Myo...

Journal: :The Journal of clinical investigation 1990
D A Greene S A Lattimer P B Carroll J D Fernstrom D N Finegold

A myo-inositol-related defect in nerve sodium-potassium ATPase activity in experimental diabetes has been suggested as a possible pathogenetic factor in diabetic neuropathy. Because the sodium-potassium ATPase is essential for other sodium-cotransport systems, and because myo-inositol-derived phosphoinositide metabolites regulate multiple membrane transport processes, sodium gradient-dependent ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2016
Gucan Dai Haijie Yu Martin Kruse Alexis Traynor-Kaplan Bertil Hille

Myo-inositol is an important cellular osmolyte in autoregulation of cell volume and fluid balance, particularly for mammalian brain and kidney cells. We find it also regulates excitability. Myo-inositol is the precursor of phosphoinositides, key signaling lipids including phosphatidylinositol 4,5-bisphosphate [PI(4,5)P2]. However, whether myo-inositol accumulation during osmoregulation affects ...

Journal: :The Journal of biological chemistry 1980
L M Hallcher W R Sherman

myo-Inositol-1-phosphatase has been partially purified from bovine brain. The enzyme has a molecular weight of about 58,000. Both L-myo-inositol 1-phosphate and D-myo-inositol 1-phosphate are hydrolyzed by the enzyme as well as (-)-chiro-inositol 3-phosphate and 2'-AMP. Triphosphoinositide is not a substrate. The phosphatase is completely dependent on Mg2+, which has a Km of 1 mM. Calcium and m...

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