نتایج جستجو برای: mitochondrial transcription factor a

تعداد نتایج: 13668788  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Yulia V Surovtseva Timothy E Shutt Justin Cotney Huseyin Cimen Sophia Y Chen Emine C Koc Gerald S Shadel

Basal transcription of human mitochondrial DNA (mtDNA) in vitro requires the single-subunit, bacteriophage-related RNA polymerase, POLRMT, and transcription factor h-mtTFB2. This two-component system is activated differentially at mtDNA promoters by human mitochondrial transcription factor A (h-mtTFA). Mitochondrial ribosomal protein L7/L12 (MRPL12) binds directly to POLRMT, but whether it does...

Journal: :iranian journal of basic medical sciences 0
fatemeh khatami department of biology, yazd university, yazd, iran mohammad mehdi heidari department of biology, yazd university, yazd, iran massoud houshmand department of medical genetics, national institute for genetic engineering and biotechnology, tehran, iran

objective(s): as mitochondrial oxidative stress is probably entailed in atp production, a candidate modifier factor for the long qt syndrome (lqts) could be mitochondrial dna (mtdna). it has been notified that ion channels' activities in cardiomyocytes are sensitive to the atp level. materials and methods: the sample of the research was an iranian family with lqts for mutations by pcr-sscp...

پایان نامه :وزارت علوم، تحقیقات و فناوری - دانشگاه علوم بهزیستی و توانبخشی 1390

اکثر مطالعات همراهی پلی مورفیسم های تک نوکلوتیدی دارای مشکل عدم تجدید پذیری هستند که این امر توالی یابی دوباره ژن ها به منظور شناسایی واریانت های نادر جدید و جهش های مضر را ضروری می سازد. در پژوهش های اخیر جهش های عملکردی در توالی پروموتری ژن کلرتیکولین ((calr در طیف بیماران سایکوز گزارش شده است که در جمعیت کنترل وجود ندارد. ژن aft6 (activating transcription factor 6)، یک فاکتور رونویسی مهم و ...

Journal: :American journal of physiology. Heart and circulatory physiology 2006
Sabzali Javadov Daniel M Purdham Asad Zeidan Morris Karmazyn

We have recently demonstrated that mitochondrial respiratory dysfunction and mitochondrial permeability transition pore opening during postinfarction remodeling are prevented by the Na(+)/H(+) exchange-1 (NHE-1)-specific inhibitor EMD-87580 (EMD). One of the mechanisms underlying the beneficial effect of NHE-1 inhibition on mitochondria could result from the drug's ability to regulate transcrip...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید