نتایج جستجو برای: lateral intake
تعداد نتایج: 230178 فیلتر نتایج به سال:
Amyotrophic lateral sclerosis, the most common adult-onset motor neuron disease, leads to death within 3 to 5 years after onset. Beyond progressive motor impairment, patients with amyotrophic lateral sclerosis suffer from major defects in energy metabolism, such as weight loss, which are well correlated with survival. Indeed, nutritional intervention targeting weight loss might improve survival...
this study was designed to examine the effects of intracerebroventricular (icv) injection of ketanserin(5-ht2a receptor antagonist) and sb242084 (5-ht2c receptor antagonist) on harmaline induced feeding anddrinking response in 24-h food-deprived (fd24) broiler cockerels. at first, guide cannula was surgicallyimplanted in the right lateral ventricle of chickens. in experiment 1, birds were injec...
Serotonin [5-hydroxytryptamine (5-HT)] and CCK injected into the lateral parabrachial nucleus (LPBN) inhibit NaCl and water intake. In this study, we investigated interactions between 5-HT and CCK into the LPBN to control water and NaCl intake. Male Holtzman rats with cannulas implanted bilaterally in the LPBN were treated with furosemide + captopril to induce water and NaCl intake. Bilateral L...
Bilateral injections of the GABA(A) agonist muscimol into the lateral parabrachial nucleus (LPBN) disrupt satiety and induce strong ingestion of water and 0.3M NaCl in fluid-replete rats by mechanisms not completely clear. In the present study, we investigated the effects of the blockade of central muscarinic cholinergic receptors with atropine injected intracerebroventricularly (i.c.v.) on 0.3...
Lesions in ventromedial part of amygdala impaired neither alimentary instrumental reflexes nor food intake, but impaired predatory behavior. Lesions localized in the more dorsal and lateral part of the amygdala produced disorders in instrumental performance and food intake without changes in predatory attack. Both kind of lesions did not impair neurological reflexes, therefore the abolishment o...
Neurochemical phenotype of hypothalamic neurons showing Fos expression 23 h after intracranial AgRP.
Agouti-related protein (AgRP) is coexpressed with neuropeptide Y (NPY) in a population of neurons in the arcuate nucleus (ARC) of the hypothalamus and stimulates food intake for up to 7 days if injected intracerebroventricularly. The prolonged food intake stimulation does not seem to depend on continued competition at the melanocortin-4 receptor (MC4R), because the relatively specific MC4R agon...
Recent evidence suggests that the central melanocortin (MC) system is a prominent contributor to food intake and body weight control. MC receptor (MC-R) populations in the arcuate and paraventricular nuclei are considered probable sites of action mediating the orexigenic effects of systemically or intracerebroventricularly administered ligands. Yet, the highest MC4-R density in the brain is fou...
Nociceptin/orphanin FQ (N/OFQ) is known to induce food intake when administered into the lateral ventricle or certain brain areas. This is somewhat contradictory to its reward-suppressing role, as food is a strong rewarding stimulus. This discrepancy may be due to the functional diversity of N/OFQ's target brain areas. N/OFQ has been shown to inhibit orexin and melanin-concentrating hormone (MC...
1 2 3 Peptide YY signaling in the lateral parabrachial nucleus increases food intake 4 through the Y1 receptor 5 6 Running Title: lPBN PYY-Y1R signaling increases food intake 7 8 Amber L. Alhadeff1, Danielle Golub1, Matthew R. Hayes2, Harvey J. Grill1 9 1Department of Psychology, 2Department of Psychiatry 10 University of Pennsylvania, Philadelphia, PA 19104 11 12 13 14 15 16 17 18 Correspondin...
Leptin reduces body fat selectively, sparing body protein. Accordingly, during chronic leptin administration, food intake is suppressed, and body weight is reduced until body fat is depleted. Body weight then stabilizes at this fat-depleted nadir, while food intake returns to normal caloric levels, presumably in defense of energy and nutritional homeostasis. This model of leptin treatment offer...
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