نتایج جستجو برای: fibroblast growth factor 2

تعداد نتایج: 3676118  

Journal: :The Journal of Cell Biology 1987
D Schubert N Ling A Baird

Heparin-binding growth factor-2 (HBGF-2; also known as basic fibroblast growth factor) is mitogenic for most anchorage-dependent cells. It is shown here that HBGF-2 stimulates cell-substratum adhesion and neurite extension in the sympathetic nerve cell line PC12. When HBGF-2 is adsorbed to artificial extracellular matrices consisting of heparin or chondroitin sulfate, it causes the formation of...

Journal: :Molecular and cellular biology 2005
Omar A Ibrahimi Brian K Yeh Anna V Eliseenkova Fuming Zhang Shaun K Olsen Makoto Igarashi Stuart A Aaronson Robert J Linhardt Moosa Mohammadi

Two competing models for fibroblast growth factor (FGF) receptor (FGFR) dimerization have recently emerged based on ternary FGF-FGFR-heparin crystal structures. In the symmetric two-end model, heparin promotes dimerization of two FGF-FGFR complexes by stabilizing bivalent interactions of the ligand and receptor through primary and secondary sites and by stabilizing direct receptor-receptor cont...

Journal: :The Journal of thoracic and cardiovascular surgery 2005
Keith A Horvath Chia Yang J Lu Emmanuel Robert Glenn F Pierce Rodney Greene Barbara A Sosnowski John Doukas

OBJECTIVES The purpose of this study was to investigate whether a novel fibroblast growth factor-2 gene formulation, providing a localized and sustained availability of the adenoviral vector from a collagen-based matrix, in combination with CO 2 transmyocardial laser revascularization would lead to an enhanced angiogenic response and improved myocardial function. METHODS Fibroblast growth fac...

Journal: :The Journal of endocrinology 2003
H Tokuda K Hirade X Wang Y Oiso O Kozawa

We previously reported that basic fibroblast growth factor (FGF-2) activates p44/p42 mitogen-activated protein (MAP) kinase resulting in the stimulation of vascular endothelial growth factor (VEGF) release in osteoblast-like MC3T3-E1 cells and that FGF-2-activated p38 MAP kinase negatively regulates VEGF release. In the present study, we investigated the involvement of stress-activated protein ...

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